Ezrin deficiency triggers glial fibrillary acidic protein upregulation and a distinct reactive astrocyte phenotype.

Schacke, Stephan; Kirkpatrick, Joanna; Stocksdale, Amy; Bauer, Reinhard; Hagel, Christian; Riecken, Lars Björn; Morrison, Helen

Schacke, Stephan; Kirkpatrick, Joanna; Stocksdale, Amy; Bauer, Reinhard; Hagel, Christian; Riecken, Lars Björn; Morrison, Helen.

Afiliação

Schacke S; Leibniz Institute on Aging, Fritz Lipmann Institute, Jena, Germany.
Kirkpatrick J; Leibniz Institute on Aging, Fritz Lipmann Institute, Jena, Germany.
Stocksdale A; Leibniz Institute on Aging, Fritz Lipmann Institute, Jena, Germany.
Bauer R; Institute of Molecular Cell Biology, CMB, Jena University Hospital, Jena, Germany.
Hagel C; Institute of Neuropathology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
Riecken LB; Leibniz Institute on Aging, Fritz Lipmann Institute, Jena, Germany.
Morrison H; Leibniz Institute on Aging, Fritz Lipmann Institute, Jena, Germany.

Glia ; 70(12): 2309-2329, 2022 12.

Article em En | MEDLINE | ID: mdl-35929192

Assuntos

Astrócitos; Gliose; Animais; Astrócitos/metabolismo; Proteínas do Citoesqueleto; Feminino; Proteína Glial Fibrilar Ácida/metabolismo; Gliose/metabolismo; Ácido Glutâmico/metabolismo; Camundongos; Camundongos Knockout; Fenótipo; Gravidez; Proteoma/metabolismo; Regulação para Cima

Palavras-chave

Ezrin; GFAP; astrocyte reactivity; peripheral astrocyte processes; proteome; reduced explorative behavior; stroke

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Astrócitos / Gliose Limite: Animals / Pregnancy Idioma: En Revista: Glia Assunto da revista: NEUROLOGIA Ano de publicação: 2022 Tipo de documento: Article País de afiliação: Alemanha

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