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Int Immunopharmacol ; 133: 112074, 2024 May 30.
Artigo em Inglês | MEDLINE | ID: mdl-38615383

RESUMO

The tumor microenvironment plays a vital role in glioblastoma growth and invasion. PD-1 and PD-L1 modulate the immunity in the brain tumor microenvironment. However, the underlying mechanisms remain unclear. In the present study, in vivo and in vitro experiments were conducted to reveal the effects of PD-1/PD-L1 on the crosstalk between microglia and glioma. Results showed that glioma cells secreted PD-L1 to the peritumoral areas, particularly microglia containing highly expressed PD-1. In the early stages of glioma, microglia mainly polarized into the pro-inflammatory subtype (M1). Subsequently, the secreted PD-L1 accumulated and bound to PD-1 on microglia, facilitating their polarization toward the microglial anti-inflammatory (M2) subtype primarily via the STAT3 signaling pathway. The role of PD-1/PD-L1 in M2 polarization of microglia was partially due to PD-1/PD-L1 depletion or application of BMS-1166, a novel inhibitor of PD-1/PD-L1. Consistently, co-culturing with microglia promoted glioma cell growth and invasion, and blocking PD-1/PD-L1 significantly suppressed these processes. Our findings reveal that the PD-1/PD-L1 axis engages in the microglial M2 polarization in the glioma microenvironment and promotes tumor growth and invasion.


Assuntos
Antígeno B7-H1 , Neoplasias Encefálicas , Glioma , Microglia , Receptor de Morte Celular Programada 1 , Animais , Humanos , Masculino , Camundongos , Antígeno B7-H1/metabolismo , Neoplasias Encefálicas/metabolismo , Neoplasias Encefálicas/patologia , Neoplasias Encefálicas/imunologia , Linhagem Celular Tumoral , Proliferação de Células/efeitos dos fármacos , Técnicas de Cocultura , Glioma/metabolismo , Glioma/patologia , Glioma/imunologia , Microglia/metabolismo , Microglia/imunologia , Receptor de Morte Celular Programada 1/metabolismo , Transdução de Sinais , Fator de Transcrição STAT3/metabolismo , Microambiente Tumoral/imunologia
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