[Traditional echography and dopplerography in the diagnosis of vascular neoplasms of the maxillofacial area in children]. / Traditsionnaia ékhografiia i dopplerografiia v diagnostike sosudistykh novoobrazovanii cheliustno-litsevoi oblasti u detei.

Twelve patients with hemangiomas of maxillofacial soft tissues aged 9 months to 15 years were examined by B-mode echography and color doppler imaging of the bloodstream. Combination of these two methods helps confirm the presence of a vascular neoformation, assess its dissemination, volume, and syntopy, specify the morphological variant of its structure, evaluate bloodstream velocity in various parts of the neoplasm. The notion of bloodstream intensity is introduced for the first time, reflecting the number of actively functioning vessels per unit of tumor volume. Causes of artefact appearance have been detected and approaches to their elimination out outlined. Echographic signs permitting selection of the optimal treatment strategy for each patient have been defined.

[Use of loading tests to detect silent myocardial ischemia]. / Primenenie nagruzochnykh prob dlia vyiavleniia bezbolevoi ishemii miokarda.

A total of 74 patients with postinfarct cardiosclerosis (PC) and stable angina (SA) were examined by Holter monitoring, bicycle ergometry, echocardiography and 201Tl chloride myocardial scintigraphy and loading tests (transesophageal cardiac pacing and isometric hand exercise test). The detection rate for silent myocardial ischemia (SI) was found to be 57.1% with Holter monitoring, 52.3% by bicycle ergometry, 62.5% with echocardiography, 100% with myocardial scintigraphy with loading test and 88.8% with that without the loading test. The detection rate for PC was 33.9, 32.0, 64.7, 81.8, and 56.7%, respectively. Higher SI detection rates in postinfarct patients were more frequently observed when echocardiography and myocardial scintigraphy in combination with loading tests in patients with PC without angina. The efficiency of SI detection in patients with various coronary heart disease increases when loading tests are employed. The loading tests in echocardiography and myocardial scintigraphy ensure the most complete detection of SI in postinfarct patients without angina.

[Painless myocardial ischemia in patients with post-infarction cardiosclerosis]. / Bezbolevaia ishemia miokarda u bol'nykh postinfarktnym kardio- sklerozom.

Holter monitoring, echocardiography and 201Tl myocardial scintigraphy were performed in 40 patients with postinfarction cardiosclerosis. The monitoring revealed silent myocardial ischemia (SMI), including 27 having SMI with concurrent anginal episodes, 7 with SMI alone. A direct correlation was found between the duration of SMI and silent myocardial infarction (r = 0.539; p less than 0.05). The patients with left ventricular failure and myocardial hypertrophy exhibited longer SMI, as evidenced by echocardiography. 201Tl myocardial scintigraphy revealed no relationship between the degree of SMI and the dimensions of a postinfarction scar. SMI was detected in 77.5% of the patients with postinfarction cardiosclerosis. The duration of SMI was longer with the presence of angina pectoris, left ventricular failure or myocardial hypertrophy.

[Comparative analysis of the levels of anti-prostaglandin F2 alpha antibodies in the normal state and in cardiovascular diseases]. / Sravnitel'nyi analiz soderzhaniia autoantitel protiv prostaglandina F2 alpha v norme i pri serdechno-sosudistykh zabolevaniiakh.

The ELISA test was performed on sera of normal donors and patients with myocardial infarction (MI) and essential hypertension (EH) to detect autoantibodies against prostaglandin F2 alpha (a-PGF2 alpha). The a-PGF2 alpha level was much higher after immune complexes dissociation. Baseline a-PGF2 alpha was elevated in EH patients with a sudden rise in blood pressure, and in MI patients without hypertension and unstable angina. MI patients with bradyarrhythmias also had elevated a-PGF2 alpha levels. Baseline a-PGF2 alpha was rather low in the sera of donors, chronic EH patients with hypertension and unstable angina, suggesting that a-PGF2 alpha has a physiological role to play.