The COVID-19 pandemic and lay perceptions of poverty and neglect.

OBJECTIVES: In cases of child neglect, intervention depends on accurate identification and reporting. Prior work has shown that individuals, especially those of high socioeconomic status (SES), conflate poverty and neglect when making identification and reporting decisions. The COVID-19 pandemic led to changes in people's experiences with poverty, likely influencing their ability to distinguish poverty in families and neglectful parenting. HYPOTHESES: Two studies tested the impact of COVID-19 on laypersons' perceptions of neglect, likelihood of reporting neglect, and attributions of blame for neglect. We hypothesized that laypersons would conflate poverty with neglect, that COVID-19 would be associated with a decreased likelihood of doing so, and that attributions of blame would mediate the latter tendency. METHOD: Adults read vignettes about a mother's care of her daughter and responded to questions about the mother's neglectfulness and their reporting likelihood. Study 1 (N = 676, Mage = 38.80, 48.08% women) compared responses collected before COVID-19 (August 2018) to responses from a separate set of adults collected during COVID-19 (November-December 2020). Study 2 (N = 704, Mage = 43.88, 63.49% women) manipulated mention of COVID-19 to assess whether cuing the pandemic affected identification and reporting, and measured attributions of blame to assess whether they explained the relation between COVID-19 and perceptions of neglect. RESULTS: Whereas most laypersons distinguished situations with versus without neglect, some conflated poverty with neglect when making identification and reporting decisions. However, COVID-19 did not have a direct impact on identification or reporting decisions. Attributions of blame partially explained laypersons' perceptions of situations as neglectful and as warranting reporting. Laypersons' current SES and perceptions of COVID-19 in 2020 were positively associated with identification and reporting. CONCLUSIONS: Laypersons in part mistake poverty for neglect, and COVID-19 had indirect effects on perceptions of neglect and reporting decisions. Public education efforts may help improve identification of vulnerable children by laypersons. (PsycInfo Database Record (c) 2022 APA, all rights reserved).

Identification and Incidence of Child Maltreatment During the COVID-19 Pandemic.

The coronavirus disease 2019 (COVID-19) pandemic has raised serious concerns about child maltreatment, which is known to increase in frequency and severity during times of high stress. The present study used diverse datasets to concurrently examine changes in identification and medical evaluation of maltreatment allegations from before to during COVID-19. Four sources of data were collected from two counties for the months of March-December in 2019 and 2020, including reports to social services and child maltreatment evaluation clinic medical evaluations (CMECs). The number of reports, number of children reported, and rate of children reported were used to evaluate identification. Incidence was estimated based on the number of medical evaluations conducted at the CMECs. Maltreatment type, reporter type, and child demographics were also considered. Across both counties, there were significantly fewer reports and reported children in 2020 compared to 2019, signifying decreased identification of suspected maltreatment cases. This was especially true in spring and fall when children are typically in school. Across both counties, the proportion of children reported to the county that received medical evaluations was higher in 2020 compared to 2019. This suggests that the pandemic was related to an increase in the occurrence maltreatment serious enough to warrant medical evaluations, or perhaps in the relative number of serious cases identified. Findings show divergent trends in reporting and evaluation of suspected maltreatment cases from before to during COVID-19. Identification and service delivery methods need creative solutions to adapt to changing environments. Medical, social, and legal systems need to prepare for increases in families seeking services as pandemic-related restrictions are lifted.

The Link Between Adversity and Dating Violence Among Adolescents Hospitalized for Psychiatric Treatment: Parental Emotion Validation as a Candidate Protective Factor.

Adolescents are at risk for becoming victims or perpetrators for a variety of forms of dating violence, including cyber violence, physical violence, psychological abuse, and sexual abuse. Interestingly, a robust predictor of dating violence is adverse experiences during childhood; however, factors that could mitigate the risk of dating violence for those exposed to adversity have seldom been examined. Using the cumulative stress hypothesis as a lens, the current study examined severity of adverse experiences as a predictor of dating violence within a sample at risk for both victimization and perpetration of dating violence: An adolescent (12-17 years old; N = 137) sample who were receiving inpatient psychiatric treatment. First, the current study aimed to replicate previous findings to determine whether adversity predicted dating violence and whether this varied by gender. Then, the current study examined one factor that could mitigate the relation between adversity and dating violence-parental emotion validation. High rates of maternal emotion validation resulted in no relation between adversity and dating violence perpetration and victimization; however, the relation was present at average and low levels of maternal emotion validation. Next, by adding gender as an additional moderator to the model, we found that high rates of paternal emotion validation extinguished the relation between adversity and dating violence perpetration, but only for adolescent boys. This pattern was not found for maternal emotion validation. Interestingly, the relation between adversity and dating violence victimization did not vary as a function of maternal or paternal validation of emotion for either child gender. These findings are discussed in terms of their meaning within this sample, possible future directions, and their implications for the prevention of dating violence.

Primary and Secondary Variants of Psychopathic Traits in at-Risk Youth: Links with Maltreatment, Aggression, and Empathy.

The current study examined whether two variants of psychopathic traits (PT) were identifiable in high-risk youth who had not yet been identified as antisocial, some of whom had documented histories of maltreatment (N = 167, Mage = 14.84), and then whether the variants differed in levels of aggression and empathy. High-PT youth with low anxiety and trauma (i.e., primary variant PT) and high anxiety and trauma (i.e., secondary variant PT) were differentiated. The secondary variant group was comprised largely of youth with documented histories of maltreatment. This group of youth also reported higher levels of proactive and reactive aggression than did the primary variant youth and low-PT youth. All youth reported similar levels of affective empathy and only small differences in cognitive empathy emerged: Primary variant youth reported lower cognitive empathy than low-PT youth. Findings support generalization of two variant groups of youth with psychopathic traits to diverse, high-risk samples not already identified as antisocial and have important implications for policy and practice.

Baseline neuropsychological profiles in prion disease predict survival time.

OBJECTIVE: Few studies have captured the neuropsychological profile of sporadic Creutzfeldt-Jakob disease (sCJD) with neuropsychological testing, and little is known about cognitive predictors of survival. We characterized baseline neuropsychological performance in sCJD and investigated associations with survival. METHODS: sCJD participants who completed the MMSE (n = 118), 61 sCJD of whom also completed a neuropsychological battery at baseline, and 135 age-matched healthy controls, were included. Composite scores of global cognition, memory, executive functions, visuospatial, and language were derived. Cox proportional hazard models estimated survival time, controlling for age and education. Additional models adjusted for Barthel Index and PRNP codon 129 polymorphism. RESULTS: sCJD participants performed significantly worse than controls on all cognitive tasks and composites with most showing very large effect sizes. The three tests showing the largest group differences were delayed verbal recall (Hedges'g = 4.08, P < 0.0001), Stroop Inhibition (Hedges'g = 3.14, P < 0.0001), and Modified Trails (Hedges'g = 2.94, P < 0.0001). Memory (95%) and executive functioning (87%) composites were most commonly impaired. Poorer global (HR = 0.65, P < 0.0001), visuospatial (HR = 0.82, P < 0.0001), and memory (HR = 0.82, P = 0.01) composites predicted shorter survival. Visuospatial cognition remained a significant predictor even after adjusting for all other cognitive composites; each standard deviation decrease in visuospatial cognition was associated with an 18% higher chance of death (HR = 0.82, P < 0.003). Global (HR = 0.68, P = 0.03) and visuospatial (HR = 0.82, P = 0.001) composites remained significant predictors after controlling for Barthel Index and codon 129. INTERPRETATION: sCJD participants exhibit a broad range of cognitive impairments, with memory and executive functioning deficits in the vast majority. Neuropsychological assessment, particularly of visuospatial abilities, informs prognostication in sCJD.

Prion disease.

Genetic prion diseases (gPrDs) are caused by autosomal-dominant mutations in the prion protein gene (PRNP). Although the first PRNP mutations identified, and most since, are PRNP missense, octapeptide repeat insertions, deletion and nonsense mutations have now also been shown to cause gPrD. Based on clinicopathologic features of familial disease, gPrDs historically have been classified into three forms: familial Jakob-Creutzfeldt disease, Gerstmann-Sträussler-Scheinker disease, and fatal familial insomnia. This classification, however, occurred prior to the identification of PRNP, and although these forms are still recognized, classification now is somewhat more complex. Clinical manifestations, and even pathology, are known to be more heterogeneous and varied than the historic three phenotypic classifications. Most gPrDs either present rapidly with progression of dementia, ataxia, myoclonus, and other motor features leading to death in few months or present more slowly, declining over a few years with mild cognitive impairment, ataxia, or parkinsonism and later dementia; a few very rare mutations, however, present over years to decades with neuropsychiatric disorders and systemic symptoms (gastrointestinal disorders and neuropathy). In this chapter, we review the broad phenotypic spectrum of PRNP mutations causing gPrDs.