RESUMO
We present a case of a patient with ataxia, dysmetria, and hemiparesis after a stroke in the corona radiata. The patient had an excellent clinical course with near resolution of symptoms in 2 and a half weeks and returned back to work full duty and full time a couple of weeks later. We use videos of several neurologic tests to demonstrate and characterize the dysmetria. Interestingly, a key characteristic of the dysmetria appears to be different from that seen in patients with dysmetria arising from a cerebellar, thalamic, or pontine lesion. We propose a possible neurophysiologic mechanism-damage to and redundancy of part of the corticopontine portion of the cerebellar circuit located in the corona radiata-respectively responsible for this condition and recovery. We also discuss how a simple noninvasive study of patients with ataxia and dysmetria secondary to corona radiata, thalamic, pontine, and possibly other brain lesions may be helpful in elucidating the contribution of pontocerebellar fibers and other structures to motor control. LEVEL OF EVIDENCE: V.
Assuntos
Ataxia Cerebelar/etiologia , Ataxia Cerebelar/reabilitação , Paresia/etiologia , Paresia/reabilitação , Acidente Vascular Cerebral/complicações , Acidente Vascular Cerebral/diagnóstico , Ataxia Cerebelar/diagnóstico , Feminino , Humanos , Pessoa de Meia-Idade , Paresia/diagnóstico , Acidente Vascular Cerebral/terapiaRESUMO
We present two cases of Amplatzer® device deployment that exhibited long-term failure necessitating surgical correction. The first case illustrates recanalization of a ruptured Valsalva aneurysm with worsening aortic insufficiency after percutaneous repair with the Amplatzer Vascular Plugs and Amplatzer duct occluder, while the second case illustrates recanalization after deployment of the Amplatzer septal occluder in a patient with a pseudoaneursym in a bovine aorta after type I aortic dissection repair. These two cases illustrate the potential long-term complications and limitations in durability with these devices.