RESUMO
A total of 192 patients with Bell paralysis were studied. In 32 a technique of biofeedback training was applied to accelerate the restoration of mimetic muscles with EMG feedback. Clinical and electrophysiological data confirmed the efficiency of this technique in terms of considerably accelerated rehabilitation.
Assuntos
Terapia por Estimulação Elétrica/métodos , Músculos Faciais/fisiopatologia , Paralisia Facial/terapia , Potenciais de Ação , Adulto , Eletromiografia , Paralisia Facial/fisiopatologia , Retroalimentação , Feminino , Humanos , Masculino , Pessoa de Meia-IdadeRESUMO
Portable, electromyographic feed-back devices were used to develop a scheme for staged and directed correction of motor function of facial muscles. The scheme was applied to the treatment of 32 patients aged 17 to 60 years with neuritis and neuropathy of the facial nerve. As a result of the treatment, the patients manifested the improvement of coordinative interrelations between the analogous groups of muscles of the diseased and normal side of the face (activation of weakened muscles with simultaneous abatement of the intensity of pathologic synergisms), evidencing the returning to normal of the central programs that control movements.
Assuntos
Biorretroalimentação Psicológica/instrumentação , Paralisia Facial/terapia , Adolescente , Adulto , Eletromiografia , Músculos Faciais/inervação , Músculos Faciais/fisiopatologia , Paralisia Facial/fisiopatologia , Feminino , Lateralidade Funcional , Humanos , Masculino , Pessoa de Meia-Idade , Contração MuscularRESUMO
A total of 230 patients with facial neuropathies were under this study. Of these, in 29 members of 13 families the hereditary nature of the diseases transmission was clearly evident. The incidence of hereditary-familial form of the facial neuropathy was 12.6% with the rate of intrafamilial affliction being 23.3%. Two factors have been found to influence the morbidity: (1) genetic predisposition manifested by autosome-dominant type of temporal bone malformations; (2) exogenous factors leading to ischemia and compression of the facial nerve and thus promoting the phenotypic realization of genotypic predisposition.