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EMBO J ; 37(20)2018 10 15.
Artigo em Inglês | MEDLINE | ID: mdl-30181118

RESUMO

The osteogenic differentiation of mesenchymal stem cells (MSCs) is governed by multiple mechanisms. Growing evidence indicates that ubiquitin-dependent protein degradation is critical for the differentiation of MSCs and bone formation; however, the function of ubiquitin-specific proteases, the largest subfamily of deubiquitylases, remains unclear. Here, we identify USP34 as a previously unknown regulator of osteogenesis. The expression of USP34 in human MSCs increases after osteogenic induction while depletion of USP34 inhibits osteogenic differentiation. Conditional knockout of Usp34 from MSCs or pre-osteoblasts leads to low bone mass in mice. Deletion of Usp34 also blunts BMP2-induced responses and impairs bone regeneration. Mechanically, we demonstrate that USP34 stabilizes both Smad1 and RUNX2 and that depletion of Smurf1 restores the osteogenic potential of Usp34-deficient MSCs in vitro Taken together, our data indicate that USP34 is required for osteogenic differentiation and bone formation.


Assuntos
Proteína Morfogenética Óssea 2/metabolismo , Diferenciação Celular , Células-Tronco Mesenquimais/metabolismo , Osteogênese , Transdução de Sinais , Proteases Específicas de Ubiquitina/metabolismo , Animais , Proteína Morfogenética Óssea 2/genética , Regeneração Óssea/genética , Técnicas de Silenciamento de Genes , Humanos , Células-Tronco Mesenquimais/citologia , Camundongos , Camundongos Knockout , Osteoblastos/citologia , Osteoblastos/metabolismo , Proteases Específicas de Ubiquitina/genética
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