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Genetic manipulation of the ApoF/Stat2 locus supports an important role for type I interferon signaling in atherosclerosis.
Lagor, William R; Fields, David W; Bauer, Robert C; Crawford, Alison; Abt, Michael C; Artis, David; Wherry, E John; Rader, Daniel J.
Afiliação
  • Lagor WR; Division of Translational Medicine and Human Genetics, Institute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA. Electronic address: lagor@bcm.edu.
  • Fields DW; Division of Translational Medicine and Human Genetics, Institute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.
  • Bauer RC; Division of Translational Medicine and Human Genetics, Institute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.
  • Crawford A; Department of Microbiology and Institute for Immunology, University of Pennsylvania, Philadelphia, PA, USA.
  • Abt MC; Department of Microbiology and Institute for Immunology, University of Pennsylvania, Philadelphia, PA, USA.
  • Artis D; Department of Microbiology and Institute for Immunology, University of Pennsylvania, Philadelphia, PA, USA.
  • Wherry EJ; Department of Microbiology and Institute for Immunology, University of Pennsylvania, Philadelphia, PA, USA.
  • Rader DJ; Division of Translational Medicine and Human Genetics, Institute for Translational Medicine and Therapeutics, Perelman School of Medicine, University of Pennsylvania, Philadelphia, PA, USA.
Atherosclerosis ; 233(1): 234-41, 2014 Mar.
Article em En | MEDLINE | ID: mdl-24529150
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apolipoproteínas / Receptores de LDL / Transdução de Sinais / Interferon Tipo I / Aterosclerose / Fator de Transcrição STAT2 Idioma: En Ano de publicação: 2014 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apolipoproteínas / Receptores de LDL / Transdução de Sinais / Interferon Tipo I / Aterosclerose / Fator de Transcrição STAT2 Idioma: En Ano de publicação: 2014 Tipo de documento: Article