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Adipose tissue insulin resistance due to loss of PI3K p110α leads to decreased energy expenditure and obesity.
Nelson, Victoria L B; Jiang, Ya-Ping; Dickman, Kathleen G; Ballou, Lisa M; Lin, Richard Z.
Afiliação
  • Nelson VL; Department of Physiology and Biophysics, Stony Brook University, Stony Brook, New York;
  • Jiang YP; Department of Physiology and Biophysics, Stony Brook University, Stony Brook, New York;
  • Dickman KG; Department of Pharmacological Sciences, Stony Brook University, Stony Brook, New York; Department of Medicine, Stony Brook University, Stony Brook, New York; and.
  • Ballou LM; Department of Physiology and Biophysics, Stony Brook University, Stony Brook, New York;
  • Lin RZ; Department of Physiology and Biophysics, Stony Brook University, Stony Brook, New York; Department of Veterans Affairs Medical Center, Northport, New York richard.lin@stonybrook.edu.
Am J Physiol Endocrinol Metab ; 306(10): E1205-16, 2014 May 15.
Article em En | MEDLINE | ID: mdl-24691033

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Resistência à Insulina / Tecido Adiposo / Metabolismo Energético / Classe I de Fosfatidilinositol 3-Quinases / Obesidade Idioma: En Ano de publicação: 2014 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Resistência à Insulina / Tecido Adiposo / Metabolismo Energético / Classe I de Fosfatidilinositol 3-Quinases / Obesidade Idioma: En Ano de publicação: 2014 Tipo de documento: Article