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BCR-ABL1 promotes leukemia by converting p27 into a cytoplasmic oncoprotein.
Agarwal, Anupriya; Mackenzie, Ryan J; Besson, Arnaud; Jeng, Sophia; Carey, Alyssa; LaTocha, Dorian H; Fleischman, Angela G; Duquesnes, Nicolas; Eide, Christopher A; Vasudevan, Kavin B; Loriaux, Marc M; Firpo, Eduardo; Cortes, Jorge E; McWeeney, Shannon; O'Hare, Thomas; Roberts, James M; Druker, Brian J; Deininger, Michael W.
Afiliação
  • Agarwal A; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Mackenzie RJ; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Besson A; Cancer Research Center of Toulouse, INSERM Unité Mixte de Recherche 1037, Université de Toulouse, Toulouse, France;
  • Jeng S; Division of Bioinformatics & Computational Biology, Knight Cancer Institute.
  • Carey A; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • LaTocha DH; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Fleischman AG; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Duquesnes N; Cancer Research Center of Toulouse, INSERM Unité Mixte de Recherche 1037, Université de Toulouse, Toulouse, France;
  • Eide CA; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR; Howard Hughes Medical Institute, and.
  • Vasudevan KB; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Loriaux MM; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR; Department of Pathology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR;
  • Firpo E; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, WA;
  • Cortes JE; Department of Leukemia, The University of Texas MD Anderson Cancer Center, Houston, TX; and.
  • McWeeney S; Division of Bioinformatics & Computational Biology, Knight Cancer Institute.
  • O'Hare T; Division of Hematology and Hematologic Malignancies and Huntsman Cancer Institute, The University of Utah, Salt Lake City, UT.
  • Roberts JM; Division of Basic Sciences, Fred Hutchinson Cancer Research Center, Seattle, WA;
  • Druker BJ; Division of Hematology and Medical Oncology, Knight Cancer Institute, Oregon Health & Science University, Portland, OR; Howard Hughes Medical Institute, and.
  • Deininger MW; Division of Hematology and Hematologic Malignancies and Huntsman Cancer Institute, The University of Utah, Salt Lake City, UT.
Blood ; 124(22): 3260-73, 2014 Nov 20.
Article em En | MEDLINE | ID: mdl-25293778
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Leucemia Mielogênica Crônica BCR-ABL Positiva / Transformação Celular Neoplásica / Proteínas de Fusão bcr-abl / Citoplasma / Inibidor de Quinase Dependente de Ciclina p27 Idioma: En Ano de publicação: 2014 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Leucemia Mielogênica Crônica BCR-ABL Positiva / Transformação Celular Neoplásica / Proteínas de Fusão bcr-abl / Citoplasma / Inibidor de Quinase Dependente de Ciclina p27 Idioma: En Ano de publicação: 2014 Tipo de documento: Article