Host response during Yersinia pestis infection of human bronchial epithelial cells involves negative regulation of autophagy and suggests a modulation of survival-related and cellular growth pathways.

Alem, Farhang; Yao, Kuan; Lane, Douglas; Calvert, Valerie; Petricoin, Emanuel F; Kramer, Liana; Hale, Martha L; Bavari, Sina; Panchal, Rekha G; Hakami, Ramin M

Alem, Farhang; Yao, Kuan; Lane, Douglas; Calvert, Valerie; Petricoin, Emanuel F; Kramer, Liana; Hale, Martha L; Bavari, Sina; Panchal, Rekha G; Hakami, Ramin M.

Afiliação

Alem F; National Center for Biodefense and Infectious Diseases and School of Systems Biology, George Mason University Manassas, VA, USA.
Yao K; National Center for Biodefense and Infectious Diseases and School of Systems Biology, George Mason University Manassas, VA, USA.
Lane D; U.S. Army Medical Research Institute of Infectious Diseases Frederick, MD, USA.
Calvert V; Center for Applied Proteomics and Molecular Medicine, School of Systems Biology, George Mason University Manassas, VA, USA.
Petricoin EF; Center for Applied Proteomics and Molecular Medicine, School of Systems Biology, George Mason University Manassas, VA, USA.
Kramer L; National Center for Biodefense and Infectious Diseases and School of Systems Biology, George Mason University Manassas, VA, USA.
Hale ML; U.S. Army Medical Research Institute of Infectious Diseases Frederick, MD, USA.
Bavari S; U.S. Army Medical Research Institute of Infectious Diseases Frederick, MD, USA.
Panchal RG; U.S. Army Medical Research Institute of Infectious Diseases Frederick, MD, USA.
Hakami RM; National Center for Biodefense and Infectious Diseases and School of Systems Biology, George Mason University Manassas, VA, USA.