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Early regression of coronary artery remodeling with esmolol and DDAH/ADMA pathway in hypertensive rats.
Quintana-Villamandos, Begoña; Arnalich-Montiel, Ana; Arribas, Silvia; Lüneburg, Nicole; Böger, Rainer H; Delgado-Martos, María Jesús; Fernández-Criado, Carmen; Delgado-Baeza, Emilio; González, María Carmen.
Afiliação
  • Quintana-Villamandos B; Department of Anesthesiology, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
  • Arnalich-Montiel A; Department of Pharmacology, Faculty of Medicine, Universidad Complutense, Madrid, Spain.
  • Arribas S; Department of Anesthesiology, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
  • Lüneburg N; Department of Physiology, Faculty of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
  • Böger RH; Department of Clinical Pharmacology and Toxicology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Delgado-Martos MJ; Department of Clinical Pharmacology and Toxicology, University Medical Center Hamburg-Eppendorf, Hamburg, Germany.
  • Fernández-Criado C; Molecular Biology Laboratory, Department of Experimental Medicine and Surgery, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
  • Delgado-Baeza E; Faculty of Medicine, Universidad Autónoma de Madrid, Madrid, Spain.
  • González MC; Molecular Biology Laboratory, Department of Experimental Medicine and Surgery, Hospital General Universitario Gregorio Marañón, Madrid, Spain.
Hypertens Res ; 39(10): 692-700, 2016 Oct.
Article em En | MEDLINE | ID: mdl-27250567

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Arginina / Propanolaminas / Vasos Coronários / Antagonistas de Receptores Adrenérgicos beta 1 / Remodelação Vascular / Amidoidrolases / Hipertensão Idioma: En Ano de publicação: 2016 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Arginina / Propanolaminas / Vasos Coronários / Antagonistas de Receptores Adrenérgicos beta 1 / Remodelação Vascular / Amidoidrolases / Hipertensão Idioma: En Ano de publicação: 2016 Tipo de documento: Article