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FOXP3+ Cells Recruited by CCL22 into Breast Cancer Correlates with Less Tumor Nodal Infiltration.
Freier, Christoph P; Kuhn, Christina; Endres, Stefan; Mayr, Doris; Friese, Klaus; Jeschke, Udo; Anz, David.
Afiliação
  • Freier CP; Department of Obstetrics and Gynecology, Ludwig Maximilians University of Munich, Munich, Germany Department of Clinical Pharmacology, Ludwig Maximilians University of Munich, Munich, Germany.
  • Kuhn C; Department of Obstetrics and Gynecology, Ludwig Maximilians University of Munich, Munich, Germany.
  • Endres S; Department of Clinical Pharmacology, Ludwig Maximilians University of Munich, Munich, Germany.
  • Mayr D; Department of Pathology, Ludwig Maximilians University of Munich, Munich, Germany.
  • Friese K; Department of Obstetrics and Gynecology, Ludwig Maximilians University of Munich, Munich, Germany.
  • Jeschke U; Department of Obstetrics and Gynecology, Ludwig Maximilians University of Munich, Munich, Germany udo.jeschke@med.uni-muenchen.de.
  • Anz D; Department of Clinical Pharmacology, Ludwig Maximilians University of Munich, Munich, Germany.
Anticancer Res ; 36(6): 3139-45, 2016 Jun.
Article em En | MEDLINE | ID: mdl-27272839
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Base de dados: MEDLINE Assunto principal: Neoplasias da Mama / Linfócitos do Interstício Tumoral / Linfócitos T Reguladores / Fatores de Transcrição Forkhead / Quimiocina CCL22 / Linfonodos Idioma: En Ano de publicação: 2016 Tipo de documento: Article
Buscar no Google
Imprimir
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PubMed Links

Base de dados: MEDLINE Assunto principal: Neoplasias da Mama / Linfócitos do Interstício Tumoral / Linfócitos T Reguladores / Fatores de Transcrição Forkhead / Quimiocina CCL22 / Linfonodos Idioma: En Ano de publicação: 2016 Tipo de documento: Article