Magel2 knockout mice manifest altered social phenotypes and a deficit in preference for social novelty.

Fountain, M D; Tao, H; Chen, C-A; Yin, J; Schaaf, C P

Fountain, M D; Tao, H; Chen, C-A; Yin, J; Schaaf, C P.

Afiliação

Fountain MD; Interdepartmental Program in Translational Biology and Molecular Medicine, Baylor College of Medicine, Houston, TX, USA.
Tao H; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.
Chen CA; Jan and Dan Duncan Neurological Research Institute, Texas Children's Hospital, Houston, TX, USA.
Yin J; Department of Molecular and Human Genetics, Baylor College of Medicine, Houston, TX, USA.
Schaaf CP; Jan and Dan Duncan Neurological Research Institute, Texas Children's Hospital, Houston, TX, USA.

Genes Brain Behav ; 16(6): 592-600, 2017 07.

Article em En | MEDLINE | ID: mdl-28296079

Assuntos

Antígenos de Neoplasias/genética; Comportamento Exploratório; Síndrome de Prader-Willi/genética; Proteínas/genética; Comportamento Social; Animais; Feminino; Masculino; Camundongos; Camundongos Endogâmicos C57BL; Fenótipo

Palavras-chave

ASD; Magel2; PWS; 15q11; Prader-Willi syndrome; Schaaf-Yang syndrome; autism; behavior; neurodevelopmental disorder; social behavior

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Síndrome de Prader-Willi / Comportamento Social / Proteínas / Comportamento Exploratório / Antígenos de Neoplasias Idioma: En Ano de publicação: 2017 Tipo de documento: Article

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