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miR-7-5p overexpression suppresses cell proliferation and promotes apoptosis through inhibiting the ability of DNA damage repair of PARP-1 and BRCA1 in TK6 cells exposed to hydroquinone.
Luo, Hao; Liang, Hairong; Chen, Yuting; Chen, Shaoyun; Xu, Yongchun; Xu, Longmei; Liu, Jiaxian; Zhou, Kairu; Peng, Jucheng; Guo, Guoqiang; Lai, Bei; Song, Li; Yang, Hui; Liu, Linhua; Peng, Jianming; Liu, Zhidong; Tang, Lin; Chen, Wen; Tang, Huanwen.
Afiliação
  • Luo H; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Liang H; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Chen Y; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Chen S; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Xu Y; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Xu L; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Liu J; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Zhou K; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Peng J; Xixiang Prevention and Health Care of Baoan, Shenzhen, China.
  • Guo G; Xixiang Prevention and Health Care of Baoan, Shenzhen, China.
  • Lai B; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Song L; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Yang H; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Liu L; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China.
  • Peng J; Huizhou Prevention and Treatment Centre for Occupational Disease, Huizhou, China.
  • Liu Z; Huizhou Prevention and Treatment Centre for Occupational Disease, Huizhou, China.
  • Tang L; School of Life Sciences, Sun Yat-sen University, Guangzhou, China.
  • Chen W; Guangzhou Key Laboratory of Environmental Pollution and Health Risk Assessment, Department of Toxicology, School of Public Health, Sun Yat-sen University, Guangzhou, China.
  • Tang H; Department of Environmental and Occupational Health, Dongguan Key Laboratory of Environmental Medicine, School of Public Health, Guangdong Medical University, Dongguan, China. Electronic address: thw@gdmu.edu.cn.
Chem Biol Interact ; 283: 84-90, 2018 Mar 01.
Article em En | MEDLINE | ID: mdl-29421518

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Proteína BRCA1 / MicroRNAs / Proliferação de Células / Reparo do DNA / Poli(ADP-Ribose) Polimerase-1 / Hidroquinonas Idioma: En Ano de publicação: 2018 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Proteína BRCA1 / MicroRNAs / Proliferação de Células / Reparo do DNA / Poli(ADP-Ribose) Polimerase-1 / Hidroquinonas Idioma: En Ano de publicação: 2018 Tipo de documento: Article