Increase in soluble protein oligomers triggers the innate immune system promoting inflammation and vascular dysfunction in the pathogenesis of sepsis.

Komic, Amel; Martinez-Quinones, Patricia; McCarthy, Cameron G; Webb, R Clinton; Wenceslau, Camilla F

Komic, Amel; Martinez-Quinones, Patricia; McCarthy, Cameron G; Webb, R Clinton; Wenceslau, Camilla F.

Afiliação

Komic A; Department of Surgery, Medical College of Georgia at Augusta University, Augusta, Georgia, U.S.A. akomic@augusta.edu Camilla.Wenceslau@Utoledo.Edu.
Martinez-Quinones P; Department of Physiology, Medical College of Georgia at Augusta University, Augusta, Georgia, U.S.A.
McCarthy CG; Department of Surgery, Medical College of Georgia at Augusta University, Augusta, Georgia, U.S.A.
Webb RC; Department of Physiology, Medical College of Georgia at Augusta University, Augusta, Georgia, U.S.A.
Wenceslau CF; Department of Physiology, Medical College of Georgia at Augusta University, Augusta, Georgia, U.S.A.

Clin Sci (Lond) ; 132(13): 1433-1438, 2018 07 18.

Article em En | MEDLINE | ID: mdl-30021912

Assuntos

Imunidade Inata; Inflamação/imunologia; Sepse/imunologia; Doenças Vasculares/imunologia; Humanos; Deficiências na Proteostase/imunologia; Receptores de Reconhecimento de Padrão/imunologia

Palavras-chave

formyl peptide receptor; inflammation; innate immunity; sepsis; soluble protein oligomer; vascular dysfunction

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Doenças Vasculares / Sepse / Imunidade Inata / Inflamação Idioma: En Ano de publicação: 2018 Tipo de documento: Article

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