Paradoxical LTP maintenance with inhibition of protein synthesis and the proteasome suggests a novel protein synthesis requirement for early LTP reversal.

Smolen, Paul; Baxter, Douglas A; Byrne, John H

Smolen, Paul; Baxter, Douglas A; Byrne, John H.

Afiliação

Smolen P; Laboratory of Origin: Department of Neurobiology and Anatomy, W. M. Keck Center for the Neurobiology of Learning and Memory, McGovern Medical School of the University of Texas Health Science Center at Houston, Houston, TX 77030, USA. Electronic address: Paul.D.Smolen@uth.tmc.edu.
Baxter DA; Laboratory of Origin: Department of Neurobiology and Anatomy, W. M. Keck Center for the Neurobiology of Learning and Memory, McGovern Medical School of the University of Texas Health Science Center at Houston, Houston, TX 77030, USA.
Byrne JH; Laboratory of Origin: Department of Neurobiology and Anatomy, W. M. Keck Center for the Neurobiology of Learning and Memory, McGovern Medical School of the University of Texas Health Science Center at Houston, Houston, TX 77030, USA.

J Theor Biol ; 457: 79-87, 2018 11 14.

Article em En | MEDLINE | ID: mdl-30138630

Assuntos

Região CA1 Hipocampal; Potenciação de Longa Duração; Modelos Neurológicos; Complexo de Endopeptidases do Proteassoma/metabolismo; Biossíntese de Proteínas; Animais; Humanos

Palavras-chave

Computational; Long-term potentiation; Proteasome; Simulation

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Biossíntese de Proteínas / Potenciação de Longa Duração / Complexo de Endopeptidases do Proteassoma / Região CA1 Hipocampal / Modelos Neurológicos Idioma: En Ano de publicação: 2018 Tipo de documento: Article

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