CXCL16 Induces the Progression of Pulmonary Fibrosis through Promoting the Phosphorylation of STAT3.

Zuo, Sheng; Zhu, Zhen; Liu, Yi; Li, Hong; Song, Shuang; Yin, Shaojun

Zuo, Sheng; Zhu, Zhen; Liu, Yi; Li, Hong; Song, Shuang; Yin, Shaojun.

Afiliação

Zuo S; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.
Zhu Z; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.
Liu Y; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.
Li H; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.
Song S; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.
Yin S; Department of Respiration, Shanghai Sixth People's Hospital East Affiliated to Shanghai University of Medicine & Health Sciences, Shanghai, China.

Can Respir J ; 2019: 2697376, 2019.

Article em En | MEDLINE | ID: mdl-31379980

Assuntos

Quimiocina CXCL16/sangue; Modelos Animais de Doenças; Fibrose Pulmonar/metabolismo; Fator de Transcrição STAT3/metabolismo; Animais; Estudos de Casos e Controles; Linhagem Celular; Progressão da Doença; Lipopolissacarídeos; Masculino; Camundongos; Fosforilação; Fator de Transcrição STAT3/antagonistas & inibidores; Tirfostinas

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Fibrose Pulmonar / Modelos Animais de Doenças / Fator de Transcrição STAT3 / Quimiocina CXCL16 Idioma: En Ano de publicação: 2019 Tipo de documento: Article

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