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Hyperhomocysteinemia: a trigger for complement-mediated TMA?
Bernards, J; Doubel, P; Meeus, G; Lerut, E; Corveleyn, A; Van Den Heuvel, L P; Meersseman, W; Kuypers, D K; Claes, K J.
Afiliação
  • Bernards J; Department of Nephrology, University Hospitals Leuven , Leuven, Belgium.
  • Doubel P; Department of Nephrology, AZ Groeninge Hospital , Kortrijk, Belgium.
  • Meeus G; Department of Nephrology, AZ Groeninge Hospital , Kortrijk, Belgium.
  • Lerut E; Department of Pathology, University Hospitals Leuven , Leuven.
  • Corveleyn A; Department of Pediatric Nephrology, Department of Development and Regeneration, University Hospitals Leuven , Leuven, Belgium.
  • Van Den Heuvel LP; Department of Pediatric Nephrology, Department of Development and Regeneration, University Hospitals Leuven , Leuven, Belgium.
  • Meersseman W; Department of Pediatric Nephrology, Radboud UMC , Nijmegen, The Netherlands.
  • Kuypers DK; Department of General Internal Medicine, University Hospitals Leuven , Leuven, Belgium.
  • Claes KJ; Department of Nephrology, University Hospitals Leuven , Leuven, Belgium.
Acta Clin Belg ; 76(1): 65-69, 2021 Feb.
Article em En | MEDLINE | ID: mdl-31401947
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Hiper-Homocisteinemia / Microangiopatias Trombóticas Idioma: En Ano de publicação: 2021 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Hiper-Homocisteinemia / Microangiopatias Trombóticas Idioma: En Ano de publicação: 2021 Tipo de documento: Article