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Mutations in CHCHD2 cause α-synuclein aggregation.
Ikeda, Aya; Nishioka, Kenya; Meng, Hongrui; Takanashi, Masashi; Hasegawa, Iwao; Inoshita, Tsuyoshi; Shiba-Fukushima, Kahori; Li, Yuanzhe; Yoshino, Hiroyo; Mori, Akio; Okuzumi, Ayami; Yamaguchi, Akihiro; Nonaka, Risa; Izawa, Nana; Ishikawa, Kei-Ichi; Saiki, Hidemoto; Morita, Masayo; Hasegawa, Masato; Hasegawa, Kazuko; Elahi, Montasir; Funayama, Manabu; Okano, Hideyuki; Akamatsu, Wado; Imai, Yuzuru; Hattori, Nobutaka.
Afiliação
  • Ikeda A; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Nishioka K; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Meng H; Department of Neurodegenerative and Demented Disorders, Juntendo University Graduate School of Medicine, Tokyo, 113-8421, Japan.
  • Takanashi M; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Hasegawa I; University Center of Legal Medicine, Kanagawa Dental University, Kanagawa 238-8580, Japan.
  • Inoshita T; Department of Treatment and Research in Multiple Sclerosis and Neuro-intractable Disease, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Shiba-Fukushima K; Department of Treatment and Research in Multiple Sclerosis and Neuro-intractable Disease, Juntendo University Graduate School of Medicine, Tokyo 113-8421, Japan.
  • Li Y; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Yoshino H; Research Institute for Diseases of Old Age, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Mori A; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Okuzumi A; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Yamaguchi A; Center for Genomic and Regenerative Medicine, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Nonaka R; Center for Genomic and Regenerative Medicine, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Izawa N; Department of Diagnosis, Prevention and Treatment of Dementia, Juntendo University Graduate School of Medicine, Tokyo, 113-8421, Japan.
  • Ishikawa KI; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Saiki H; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Morita M; Center for Genomic and Regenerative Medicine, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Hasegawa M; Department of Neurology, Tazuke Kofukai Medical Research Institute and Kitano Hospital, Osaka 530-8480, Japan.
  • Hasegawa K; Department of Neurology, Jikei University Katsushika Medical Center, Tokyo 125-8506, Japan.
  • Elahi M; Department of Dementia and Higher Brain Function, Tokyo Metropolitan Institute of Medical Science, Tokyo 156-8506, Japan.
  • Funayama M; Department of Neurology, NHO Sagamihara National Hospital, Kanagawa 252-0392, Japan.
  • Okano H; Department of Diagnosis, Prevention and Treatment of Dementia, Juntendo University Graduate School of Medicine, Tokyo, 113-8421, Japan.
  • Akamatsu W; Department of Neurology, Juntendo University School of Medicine, Tokyo 113-8421, Japan.
  • Imai Y; Research Institute for Diseases of Old Age, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
  • Hattori N; Center for Genomic and Regenerative Medicine, Graduate School of Medicine, Juntendo University, Tokyo 113-8421, Japan.
Hum Mol Genet ; 28(23): 3895-3911, 2019 12 01.
Article em En | MEDLINE | ID: mdl-31600778
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Doença de Parkinson / Fatores de Transcrição / Proteínas de Ligação a DNA / Alfa-Sinucleína / Mutação com Perda de Função Idioma: En Ano de publicação: 2019 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Doença de Parkinson / Fatores de Transcrição / Proteínas de Ligação a DNA / Alfa-Sinucleína / Mutação com Perda de Função Idioma: En Ano de publicação: 2019 Tipo de documento: Article