Abnormal overexpression of G9a in melanoma cells promotes cancer progression via upregulation of the Notch1 signaling pathway.

Dang, Ning-Ning; Jiao, Jing; Meng, Xianguang; An, Yunhe; Han, Chen; Huang, Shuhong

Dang, Ning-Ning; Jiao, Jing; Meng, Xianguang; An, Yunhe; Han, Chen; Huang, Shuhong.

Afiliação

Dang NN; Department of Dermatology, Jinan Central Hospital Affiliated to Shandong University, Jinan, Shandong Province, China.
Jiao J; Department of Dermatology, Jinan Central Hospital Affiliated to Shandong University, Jinan, Shandong Province, China.
Meng X; Department of Dermatology, Jinan Central Hospital Affiliated to Shandong University, Jinan, Shandong Province, China.
An Y; Beijing Center for Physical and Chemical Analysis, Beijing, China.
Han C; Institute of Basic Medicine, The First Affiliated Hospital of Shandong First Medical University, Jinan, Shandong Province, China.
Huang S; Institute of Basic Medicine, The First Affiliated Hospital of Shandong First Medical University, Jinan, Shandong Province, China.

Aging (Albany NY) ; 12(3): 2393-2407, 2020 02 03.

Article em En | MEDLINE | ID: mdl-32015216

Assuntos

Biomarcadores Tumorais/metabolismo; Antígenos de Histocompatibilidade/metabolismo; Histona-Lisina N-Metiltransferase/metabolismo; Melanoma/patologia; Receptor Notch1/metabolismo; Neoplasias Cutâneas/patologia; Animais; Biomarcadores Tumorais/análise; Progressão da Doença; Feminino; Xenoenxertos; Humanos; Masculino; Camundongos; Transdução de Sinais/fisiologia; Regulação para Cima; Melanoma Maligno Cutâneo

Palavras-chave

G9a; Notch1; UNC0642; melanoma; proliferation

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Neoplasias Cutâneas / Biomarcadores Tumorais / Histona-Lisina N-Metiltransferase / Receptor Notch1 / Antígenos de Histocompatibilidade / Melanoma Idioma: En Ano de publicação: 2020 Tipo de documento: Article

Texto completo

Imprimir

XML

PubMed Links

Buscar no Google