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Thiamine treatment preserves cardiac function against ischemia injury via maintaining mitochondrial size and ATP levels.
Yamada, Yuki; Kusakari, Yoichiro; Akaoka, Munetoshi; Watanabe, Masato; Tanihata, Jun; Nishioka, Naritomo; Bochimoto, Hiroki; Akaike, Toru; Tachibana, Toshiaki; Minamisawa, Susumu.
Afiliação
  • Yamada Y; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Kusakari Y; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Akaoka M; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Watanabe M; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Tanihata J; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Nishioka N; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Bochimoto H; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Akaike T; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
  • Tachibana T; Division of Molecular Cell Biology, Core Research Facilities for Basic Science, The Jikei University School of Medicine, Tokyo, Japan.
  • Minamisawa S; Department of Cell Physiology, The Jikei University School of Medicine, Tokyo, Japan.
J Appl Physiol (1985) ; 130(1): 26-35, 2021 01 01.
Article em En | MEDLINE | ID: mdl-33119470
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Trifosfato de Adenosina / Isquemia Miocárdica Idioma: En Ano de publicação: 2021 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Trifosfato de Adenosina / Isquemia Miocárdica Idioma: En Ano de publicação: 2021 Tipo de documento: Article