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Loss of all three APP family members during development impairs synaptic function and plasticity, disrupts learning, and causes an autism-like phenotype.
Steubler, Vicky; Erdinger, Susanne; Back, Michaela K; Ludewig, Susann; Fässler, Dominique; Richter, Max; Han, Kang; Slomianka, Lutz; Amrein, Irmgard; von Engelhardt, Jakob; Wolfer, David P; Korte, Martin; Müller, Ulrike C.
Afiliação
  • Steubler V; Department of Functional Genomics, Institute of Pharmacy and Molecular Biotechnology, Heidelberg University, Heidelberg, Germany.
  • Erdinger S; Department of Functional Genomics, Institute of Pharmacy and Molecular Biotechnology, Heidelberg University, Heidelberg, Germany.
  • Back MK; Institute of Pathophysiology, Focus Program Translational Neuroscience (FTN), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
  • Ludewig S; Division of Cellular Neurobiology, Zoological Institute, TU Braunschweig, Braunschweig, Germany.
  • Fässler D; Helmholtz Centre for Infection Research, Neuroinflammation and Neurodegeneration Group, Braunschweig, Germany.
  • Richter M; Department of Functional Genomics, Institute of Pharmacy and Molecular Biotechnology, Heidelberg University, Heidelberg, Germany.
  • Han K; Department of Functional Genomics, Institute of Pharmacy and Molecular Biotechnology, Heidelberg University, Heidelberg, Germany.
  • Slomianka L; Department of Functional Genomics, Institute of Pharmacy and Molecular Biotechnology, Heidelberg University, Heidelberg, Germany.
  • Amrein I; Institute of Anatomy and Zurich Center for Integrative Human Physiology, University of Zurich, Zurich, Switzerland.
  • von Engelhardt J; Institute of Anatomy and Zurich Center for Integrative Human Physiology, University of Zurich, Zurich, Switzerland.
  • Wolfer DP; Institute of Pathophysiology, Focus Program Translational Neuroscience (FTN), University Medical Center of the Johannes Gutenberg University Mainz, Mainz, Germany.
  • Korte M; Institute of Anatomy and Zurich Center for Integrative Human Physiology, University of Zurich, Zurich, Switzerland.
  • Müller UC; Institute of Human Movement Sciences, ETH Zurich, Zurich, Switzerland.
EMBO J ; 40(12): e107471, 2021 06 15.
Article em En | MEDLINE | ID: mdl-34008862
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Transtorno Autístico / Precursor de Proteína beta-Amiloide Idioma: En Ano de publicação: 2021 Tipo de documento: Article
Texto completo
Imprimir
XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Transtorno Autístico / Precursor de Proteína beta-Amiloide Idioma: En Ano de publicação: 2021 Tipo de documento: Article