Mito-Mendelian interactions alter in vivo glucose metabolism and insulin sensitivity in healthy mice.

Sammy, Melissa J; Connelly, Ashley W; Brown, Jamelle A; Holleman, Cassie; Habegger, Kirk M; Ballinger, Scott W

Sammy, Melissa J; Connelly, Ashley W; Brown, Jamelle A; Holleman, Cassie; Habegger, Kirk M; Ballinger, Scott W.

Afiliação

Sammy MJ; Division of Molecular and Cellular Pathology, Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama.
Connelly AW; Center for Free Radical Biology, University of Alabama at Birmingham, Birmingham, Alabama.
Brown JA; Division of Molecular and Cellular Pathology, Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama.
Holleman C; Division of Molecular and Cellular Pathology, Department of Pathology, University of Alabama at Birmingham, Birmingham, Alabama.
Habegger KM; Center for Free Radical Biology, University of Alabama at Birmingham, Birmingham, Alabama.
Ballinger SW; Division of Endocrinology, Diabetes & Metabolism, Department of Medicine, University of Alabama at Birmingham, Birmingham, Alabama.

Am J Physiol Endocrinol Metab ; 321(4): E521-E529, 2021 10 01.

Article em En | MEDLINE | ID: mdl-34370595

Assuntos

DNA Mitocondrial/metabolismo; Glucose/metabolismo; Resistência à Insulina; Insulina/metabolismo; Mitocôndrias/metabolismo; Animais; DNA Mitocondrial/genética; Feminino; Masculino; Análise da Randomização Mendeliana; Camundongos; Camundongos Endogâmicos C3H; Camundongos Endogâmicos C57BL

Palavras-chave

adiposity; glucose metabolism; insulin sensitivity; mitochondrial DNA; mitochondrial-nuclear eXchange

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Texto completo: 1 Base de dados: MEDLINE Assunto principal: DNA Mitocondrial / Resistência à Insulina / Glucose / Insulina / Mitocôndrias Idioma: En Ano de publicação: 2021 Tipo de documento: Article

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