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CRL2-KLHDC3 E3 ubiquitin ligase complex suppresses ferroptosis through promoting p14ARF degradation.
Zhang, Pingzhao; Gao, Kun; Zhang, Liang; Sun, Huiru; Zhao, Xiaying; Liu, Yajuan; Lv, Zeheng; Shi, Qing; Chen, Yingji; Jiao, Dongyue; Li, Yao; Gu, Wei; Wang, Chenji.
Afiliação
  • Zhang P; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China. pzzhang@fudan.edu.cn.
  • Gao K; Institute for Cancer Genetics, College of Physicians and Surgeons, Columbia University, New York, NY, USA. pzzhang@fudan.edu.cn.
  • Zhang L; Department of Clinical Laboratory, Shanghai First Maternity and Infant Hospital, School of Medicine, Tongji University, Shanghai, China. kungao@tongji.edu.cn.
  • Sun H; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Zhao X; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Liu Y; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Lv Z; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Shi Q; Department of Clinical Laboratory, Shanghai First Maternity and Infant Hospital, School of Medicine, Tongji University, Shanghai, China.
  • Chen Y; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Jiao D; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Li Y; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Gu W; Department of Pathology, School of Basic Medical Sciences, Fudan University Shanghai Cancer Center, State Key Laboratory of Genetic Engineering, MOE Engineering Research Center of Gene Technology, School of Life Sciences, Fudan University, Shanghai, China.
  • Wang C; Institute for Cancer Genetics, College of Physicians and Surgeons, Columbia University, New York, NY, USA. wg8@cumc.columbia.edu.
Cell Death Differ ; 29(4): 758-771, 2022 04.
Article em En | MEDLINE | ID: mdl-34743205
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas de Ciclo Celular / Proteína Supressora de Tumor p14ARF / Ubiquitina-Proteína Ligases / Ferroptose Idioma: En Ano de publicação: 2022 Tipo de documento: Article
Texto completo
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteínas de Ciclo Celular / Proteína Supressora de Tumor p14ARF / Ubiquitina-Proteína Ligases / Ferroptose Idioma: En Ano de publicação: 2022 Tipo de documento: Article