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Latexin deletion protects against radiation-induced hematopoietic damages via selective activation of Bcl-2 prosurvival pathway.
Zhang, Cuiping; Cui, Xiaojing; Liu, Yi; Wang, Fang; Signer, Robert; Nattamai, Kapana; Zhou, Daohong; Zheng, Yi; Geiger, Hartmut; Wan, Fengyi; Liang, Ying.
Afiliação
  • Zhang C; Department of Toxicology and Cancer Biology.
  • Cui X; Department of Toxicology and Cancer Biology.
  • Liu Y; Department of Physiology, University of Kentucky, Lexington, KY.
  • Wang F; Department of Toxicology and Cancer Biology.
  • Signer R; Division of Regenerative Medicine, Department of Medicine, Moores Cancer Center, University of California San Diego, La Jolla, CA.
  • Nattamai K; Experimental Hematology and Cancer Biology, Cincinnati Children Hospital Medical Center, University of Cincinnati, OH.
  • Zhou D; Department of Biochemistry and Structural Biology, University of Texas Health Science Center, San Antonio, TX.
  • Zheng Y; Experimental Hematology and Cancer Biology, Cincinnati Children Hospital Medical Center, University of Cincinnati, OH.
  • Geiger H; Institute of Molecular Medicine, Ulm University, Meyerhofstrasse, Ulm.
  • Wan F; Department of Biochemistry and Molecular Biology, Bloomberg School of Public Health, Johns Hopkins University, Baltimore, MD.
  • Liang Y; Department of Toxicology and Cancer Biology. ying.liang@uky.edu.
Haematologica ; 108(12): 3464-3470, 2023 Dec 01.
Article em En | MEDLINE | ID: mdl-37345464
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose Idioma: En Ano de publicação: 2023 Tipo de documento: Article
Texto completo
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PubMed Links
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose Idioma: En Ano de publicação: 2023 Tipo de documento: Article