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CD95 (Fas) and CD95L (FasL)-mediated non-canonical signaling pathways.
Haymour, Layla; Jean, Mickael; Smulski, Cristian; Legembre, Patrick.
Afiliação
  • Haymour L; UMR CNRS 7276, INSERM U1262, CRIBL, Université Limoges, Limoges, France.
  • Jean M; Université de Rennes, Institut des Sciences Chimiques de Rennes - UMR CNRS 6226 Equipe COrInt, Rennes F-35000, France.
  • Smulski C; Medical Physics Department, Centro Atómico Bariloche, Comisión Nacional de Energía Atómica (CNEA), Consejo Nacional de Investigaciones Científicas y Técnicas (CONICET), Río Negro, Argentina.
  • Legembre P; UMR CNRS 7276, INSERM U1262, CRIBL, Université Limoges, Limoges, France. Electronic address: patrick.legembre@inserm.fr.
Biochim Biophys Acta Rev Cancer ; 1878(6): 189004, 2023 11.
Article em En | MEDLINE | ID: mdl-37865305
Although the interaction of CD95L (also known as FasL) with its so-called death receptor CD95 (Fas) induces an apoptotic signal responsible for the elimination of infected and cancer cells and maintenance of tissue homeostasis, this receptor can also implement non apoptotic signaling pathways. This latter signaling is involved in metastatic dissemination in certain cancers and the severity of auto-immune disorders. The signaling complexity of this pair is increased by the fact that CD95 expression itself seems to contribute to oncogenesis via a CD95L-independent manner and, that both ligand and receptor might interact with other partners modulating their pathophysiological functions. Finally, CD95L itself can trigger cell signaling in immune cells rendering complex the interpretation of mouse models in which CD95 or CD95L are knocked out. Herein, we discuss these non-canonical responses and their biological functions.
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Neoplasias Idioma: En Ano de publicação: 2023 Tipo de documento: Article

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Apoptose / Neoplasias Idioma: En Ano de publicação: 2023 Tipo de documento: Article