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17ß-Estradiol mediates TGFBR3/Smad2/3 signaling to attenuate the fibrosis of TGF-ß1-induced bovine endometrial epithelial cells via GPER.
Fang, Zihao; Wang, Qian; Duan, Hongwei; Sheng, Xihui; Qi, Xiaolong; Xing, Kai; Liu, Bingying; Chang, Di; Guo, Yong; Wang, Xiangguo; Xiao, Longfei.
Afiliação
  • Fang Z; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Wang Q; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Duan H; Beijing Municipal Changping district Bureau of Agriculture and Rural Affairs, Beijing, China.
  • Sheng X; College of Veterinary Medicine, Gansu Agricultural University, Lanzhou, Gansu, China.
  • Qi X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Xing K; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Liu B; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Chang D; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Guo Y; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Wang X; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
  • Xiao L; Animal Science and Technology College, Beijing University of Agriculture, Beijing, China.
J Cell Physiol ; 239(1): 166-179, 2024 Jan.
Article em En | MEDLINE | ID: mdl-37991438
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Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteoglicanas / Receptores de Fatores de Crescimento Transformadores beta / Endometrite / Estradiol / Fator de Crescimento Transformador beta1 Idioma: En Ano de publicação: 2024 Tipo de documento: Article
Texto completo
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XML
PubMed Links
Buscar no Google

Texto completo: 1 Base de dados: MEDLINE Assunto principal: Proteoglicanas / Receptores de Fatores de Crescimento Transformadores beta / Endometrite / Estradiol / Fator de Crescimento Transformador beta1 Idioma: En Ano de publicação: 2024 Tipo de documento: Article