RESUMEN
We establish the propagation model of orbital angular momentum (OAM) modes carried by hollow vortex Gaussian (hvG) beams propagating in anisotropic atmospheric turbulence. Effects of light source parameters and atmospheric conditions on the OAM mode propagation performance are investigated in detail. The findings indicate the hvG beam with a smaller OAM quantum number, a larger beam order, or a longer source wavelength has more robust resistance to atmospheric turbulence interference. The waist width of the light source has different influences on the OAM mode propagation at different propagation distances. Atmospheric turbulences with larger values of anisotropy, inner-scale factor, non-Kolmogorov power spectrum index, and altitude are favorable for the OAM mode propagation. These research results are conducive to optimizing the design of light sources and space wireless communication systems with hvG beams.
RESUMEN
The propagation model of orbital angular momentum (OAM) modes carried by the perfect vortex (pv) beam through anisotropic oceanic turbulence links is established and the factors influencing the OAM propagation are discussed. The findings show that the self-focusing property of pv beams is beneficial to the propagation of OAM modes: a smaller topological charge, a smaller initial radius, and an optimized half-ring width can alleviate degrading effects of turbulence on the pv beam. Additionally, the pv beam with a longer wavelength is more resistant to turbulent interference. The oceanic conditions with a higher dissipation rate of kinetic energy per unit mass of seawater, larger values of anisotropy and inner-scale factor, a smaller temperature-salinity contribution ratio, or a lower mean-squared temperature dissipation rate can improve the signal mode detection probability. The results are expected to further optimize the design of OAM-based underwater wireless communication systems.
RESUMEN
We present what is, to the best of our knowledge, the first pure-three-level Yb:Ca4GdO(BO3)3 (Yb:GdCOB) laser emitting at 976 nm based on the F5/22-F27/2 transition, generally used for a quasi-three-level emission at 1032 nm. A maximum power of 782 mW at 976 nm has been achieved in continuous-wave (CW) operation pumped by a quasi-three-level Nd:SrLaAlO4 laser emitting at 902 nm. Moreover, a self-frequency-doubling CW blue laser has also been demonstrated with a maximum power of 133 mW at 488 nm.
RESUMEN
Atherosclerosis is a cardiovascular disease, which is characterized by the interaction between carbohydrates, lipids, cells and various other molecules and genetic factors. Previous studies have demonstrated that resveratrol (RV) served protective roles in numerous types of human disease by regulating different signaling pathways. The aim of the present study was to investigate the therapeutic effects of RV and analyze the potential RV-mediated mechanism in umbilical vein endothelial cells (UVECS) in atherosclerosis model mice. Reverse transcription-quantitative PCR, western blotting and immunohistochemistry were used to analyze the therapeutic effects of RV both in vitro and in vivo. The results demonstrated that total cholesterol, triglycerides, low-density lipoprotein cholesterin and high-density lipoprotein cholesterin levels were significantly decreased in the RV group compared with the control group. RV demonstrated significant anti-atherosclerotic activity, which was determined through the atherogenic index, 3-hydroxy-3-methyl-glutaryl-Coa (HMG-CoA) reductase activity and marker enzymes, such as lactate dehydrogenase, creatine phosphokinase, aspartate transaminase, alanine transaminase and alkaline phosphatase. It was also observed that RV treatment significantly decreased the area of the arteriosclerotic lesion in the RV group compared with the control, as well as significantly decreasing the expression levels of matrix metalloproteinase-9 and CD40 ligand (CD40L) in arterial lesion tissue compared with the control group. Serum expression levels of tumor necrosis factor-α and C-reactive protein were also significantly decreased by RV treatment compared with the control group. Furthermore, RV treatment significantly decreased the expression levels of PI3K, AKT and mTOR in UVECS in vitro. In conclusion, these results suggested that the anti-atherosclerotic activity of RV may be due to its modulatory activity over the PI3K/AKT/mTOR signaling pathway. These findings suggested a potential novel treatment option for patients with atherosclerosis.