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Eur Rev Med Pharmacol Sci ; 19(4): 545-51, 2015 Feb.
Artículo en Inglés | MEDLINE | ID: mdl-25753868

RESUMEN

OBJECTIVE: The chondrocytes, the resident cells of cartilage, are maintained and take effects in the whole life upon chronic hypoxic exposure, which hypoxia-inducible factor 1 alpha (HIF-1α) play pivotal roles in response to. Dysregulation of some microRNA (miRNAs) have also been identified to be involved in hypoxia-related physiologic and pathophysiologic responses in some tissues or cell lines. However, the mechanism of miRNAs reponse to hypoxia remain largely unknown in chondrocytes, including the microRNA-195 (miR-195). AIM To investigate the effects of microRNAs (miRNAs) and hypoxia-inducible factor 1 alpha (HIF-1α) on chondrocytes in physiologic environment. MATERIALS AND METHODS: We compared the expression of miR-195 and HIF-1α mRNA on hypoxia with that on normoxia in ATDC 5 cells by qRT-PCR. Further experiments was performed to confirmed the relationships of miR-195 and HIF-1α by bioinformatics analysis and dual reporter gene assay. we also assessed the effect of miR-195 on apoptosis in hypoxic ATDC 5 cells by transfect with miR-195 mimics. RESULTS: It was found the downregulated miR-195 and upregulated HIF-1α were present in hypoxic ATDC 5 cells. miR-195 negatively regulated HIF-1α by targeting its 3'-untranslated region. Moreover, the founding indicated miR-195 greatly increased apoptosis and downregulated HIF-1α mRNA occurred simultaneously in hypoxic chondrocytes. CONCLUSIONS: We concluded that miR-195 induced apoptosis in hypoxic chondrocytes by directly targeting HIF-1α.


Asunto(s)
Apoptosis/genética , Condrocitos/fisiología , Subunidad alfa del Factor 1 Inducible por Hipoxia/genética , MicroARNs/fisiología , Hipoxia de la Célula/genética , Células Cultivadas , Condrocitos/metabolismo , Regulación de la Expresión Génica , Células HEK293 , Humanos , Regulación hacia Arriba
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