Detalhe da pesquisa
1.
CD11c+CD88+CD317+ myeloid cells are critical mediators of persistent CNS autoimmunity.
Proc Natl Acad Sci U S A
; 118(14)2021 04 06.
Artigo
em Inglês
| MEDLINE | ID: mdl-33785592
2.
Pathways leading to an immunological disease: systemic lupus erythematosus.
Rheumatology (Oxford)
; 56(suppl_1): i55-i66, 2017 04 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-28375453
3.
Behavioural and neurological symptoms accompanied by cellular neuroinflammation in IL-10-deficient mice infected with Plasmodium chabaudi.
Malar J
; 15(1): 428, 2016 08 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-27557867
4.
Developmental maturation of innate immune cell function correlates with susceptibility to central nervous system autoimmunity.
Eur J Immunol
; 43(8): 2078-88, 2013 Aug.
Artigo
em Inglês
| MEDLINE | ID: mdl-23637087
5.
The neonatal CNS is not conducive for encephalitogenic Th1 T cells and B cells during experimental autoimmune encephalomyelitis.
J Neuroinflammation
; 10: 67, 2013 May 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-23705890
6.
CCL2 upregulation triggers hypoxic preconditioning-induced protection from stroke.
J Neuroinflammation
; 9: 33, 2012 Feb 16.
Artigo
em Inglês
| MEDLINE | ID: mdl-22340958
7.
Memory B cells from a subset of treatment-naïve relapsing-remitting multiple sclerosis patients elicit CD4(+) T-cell proliferation and IFN-γ production in response to myelin basic protein and myelin oligodendrocyte glycoprotein.
Eur J Immunol
; 40(10): 2942-56, 2010 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-20812237
8.
Lymph node-derived donor encephalitogenic CD4+ T cells in C57BL/6 mice adoptive transfer experimental autoimmune encephalomyelitis highly express GM-CSF and T-bet.
J Neuroinflammation
; 8: 73, 2011 Jun 24.
Artigo
em Inglês
| MEDLINE | ID: mdl-21702922
9.
Silencing Nogo-A promotes functional recovery in demyelinating disease.
Ann Neurol
; 67(4): 498-507, 2010 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-20437585
10.
Pharmacological prion protein silencing accelerates central nervous system autoimmune disease via T cell receptor signalling.
Brain
; 133(Pt 2): 375-88, 2010 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-20145049
11.
Limitations of cell-lineage-specific non-dynamic gene recombination in CD11c.Cre+ITGA4fl/fl mice.
J Neuroimmunol
; 344: 577245, 2020 07 15.
Artigo
em Inglês
| MEDLINE | ID: mdl-32335319
12.
α4-integrin deficiency in B cells does not affect disease in a T-cell-mediated EAE disease model.
Neurol Neuroimmunol Neuroinflamm
; 6(4): e563, 2019 07.
Artigo
em Inglês
| MEDLINE | ID: mdl-31086806
13.
Defining standard enzymatic dissociation methods for individual brains and spinal cords in EAE.
Neurol Neuroimmunol Neuroinflamm
; 5(2): e437, 2018 Mar.
Artigo
em Inglês
| MEDLINE | ID: mdl-29359175
14.
Potential risk of progressive multifocal leukoencephalopathy with natalizumab therapy: possible interventions.
Arch Neurol
; 64(2): 169-76, 2007 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-17296831
15.
Altered CD4+/CD8+ T-cell ratios in cerebrospinal fluid of natalizumab-treated patients with multiple sclerosis.
Arch Neurol
; 63(10): 1383-7, 2006 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-17030653
16.
IL-12/IL-23p40 Is Highly Expressed in Secondary Lymphoid Organs and the CNS during All Stages of EAE, but Its Deletion Does Not Affect Disease Perpetuation.
PLoS One
; 11(10): e0165248, 2016.
Artigo
em Inglês
| MEDLINE | ID: mdl-27780253
17.
Effect of rituximab on the peripheral blood and cerebrospinal fluid B cells in patients with primary progressive multiple sclerosis.
Arch Neurol
; 62(2): 258-64, 2005 Feb.
Artigo
em Inglês
| MEDLINE | ID: mdl-15710854
18.
DNA plasmid vaccination for multiple sclerosis.
Arch Neurol
; 64(10): 1385-6, 2007 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-17923622
19.
High incidence of post-lumbar puncture headaches in patients with multiple sclerosis treated with natalizumab: role of intrathecal leukocytes.
Arch Neurol
; 64(7): 1055-6, 2007 Jul.
Artigo
em Inglês
| MEDLINE | ID: mdl-17620501
20.
Therapeutic potential of small interfering RNA for central nervous system diseases.
Arch Neurol
; 62(12): 1810-3, 2005 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-16344338