Detalhe da pesquisa
1.
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies.
J Mol Cell Cardiol
; 178: 22-35, 2023 05.
Artigo
em Inglês
| MEDLINE | ID: mdl-36948385
2.
Activation of Peroxisome Proliferator-Activated Receptor-δ as Novel Therapeutic Strategy to Prevent In-Stent Restenosis and Stent Thrombosis.
Arterioscler Thromb Vasc Biol
; 36(8): 1534-48, 2016 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-27283742
3.
Embryonic cardiomyocytes can orchestrate various cell protective mechanisms to survive mitochondrial stress.
J Mol Cell Cardiol
; 97: 1-14, 2016 08.
Artigo
em Inglês
| MEDLINE | ID: mdl-27106802
4.
Fine mapping of the 1p36 deletion syndrome identifies mutation of PRDM16 as a cause of cardiomyopathy.
Am J Hum Genet
; 93(1): 67-77, 2013 Jul 11.
Artigo
em Inglês
| MEDLINE | ID: mdl-23768516
5.
Cardiomyocyte maturation alters molecular stress response capacities and determines cell survival upon mitochondrial dysfunction.
Free Radic Biol Med
; 213: 248-265, 2024 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-38266827
6.
Inhibition of mitochondrial respiration has fundamentally different effects on proliferation, cell survival and stress response in immature versus differentiated cardiomyocyte cell lines.
Front Cell Dev Biol
; 10: 1011639, 2022.
Artigo
em Inglês
| MEDLINE | ID: mdl-36211452
7.
Arrhythmogenic right ventricular cardiomyopathy type 5 is a fully penetrant, lethal arrhythmic disorder caused by a missense mutation in the TMEM43 gene.
Am J Hum Genet
; 82(4): 809-21, 2008 Apr.
Artigo
em Inglês
| MEDLINE | ID: mdl-18313022
8.
Growth plasticity of the embryonic and fetal heart.
Bioessays
; 31(12): 1288-98, 2009 Dec.
Artigo
em Inglês
| MEDLINE | ID: mdl-19921657
9.
Mutations in sarcomere protein genes in left ventricular noncompaction.
Circulation
; 117(22): 2893-901, 2008 Jun 03.
Artigo
em Inglês
| MEDLINE | ID: mdl-18506004
10.
Dietary protein restriction throughout intrauterine and postnatal life results in potentially beneficial myocardial tissue remodeling in the adult mouse heart.
Sci Rep
; 9(1): 15126, 2019 10 22.
Artigo
em Inglês
| MEDLINE | ID: mdl-31641210
11.
Prenatal Mechanistic Target of Rapamycin Complex 1 (m TORC1) Inhibition by Rapamycin Treatment of Pregnant Mice Causes Intrauterine Growth Restriction and Alters Postnatal Cardiac Growth, Morphology, and Function.
J Am Heart Assoc
; 6(8)2017 Aug 04.
Artigo
em Inglês
| MEDLINE | ID: mdl-28778941
12.
Transcriptional profiling of regenerating embryonic mouse hearts.
Genom Data
; 9: 145-7, 2016 Sep.
Artigo
em Inglês
| MEDLINE | ID: mdl-27583204
13.
Impaired myocardial development resulting in neonatal cardiac hypoplasia alters postnatal growth and stress response in the heart.
Cardiovasc Res
; 106(1): 43-54, 2015 Apr 01.
Artigo
em Inglês
| MEDLINE | ID: mdl-25661081
14.
Heart development: mitochondria in command of cardiomyocyte differentiation.
Dev Cell
; 21(3): 392-3, 2011 Sep 13.
Artigo
em Inglês
| MEDLINE | ID: mdl-21920309
15.
Compensatory growth of healthy cardiac cells in the presence of diseased cells restores tissue homeostasis during heart development.
Dev Cell
; 15(4): 521-33, 2008 Oct.
Artigo
em Inglês
| MEDLINE | ID: mdl-18854137