Heterogeneous expression of Gap junction channels in the heart leads to conduction defects and ventricular dysfunction.

Gutstein, D E; Morley, G E; Vaidya, D; Liu, F; Chen, F L; Stuhlmann, H; Fishman, G I

Gutstein, D E; Morley, G E; Vaidya, D; Liu, F; Chen, F L; Stuhlmann, H; Fishman, G I.

Affiliation

Gutstein DE; Section of Myocardial Biology, Department of Medicine, Mount Sinai School of Medicine, New York, NY, USA.

Circulation ; 104(10): 1194-9, 2001 Sep 04.

Article in En | MEDLINE | ID: mdl-11535579

Subject(s)

Connexin 43/metabolism; Heart Conduction System/physiopathology; Heart Ventricles/physiopathology; Animals; Arrhythmias, Cardiac/genetics; Arrhythmias, Cardiac/physiopathology; Blotting, Western; Connexin 43/genetics; Echocardiography; Gene Expression; Genotype; Green Fluorescent Proteins; Heart Conduction System/metabolism; Heart Ventricles/metabolism; Luminescent Proteins/genetics; Luminescent Proteins/metabolism; Mice; Mice, Inbred C57BL; Mice, Inbred Strains; Mice, Knockout; Myocardial Contraction/genetics; Myocardial Contraction/physiology; Recombinant Fusion Proteins/genetics; Recombinant Fusion Proteins/metabolism; Tissue Distribution; beta-Galactosidase/genetics; beta-Galactosidase/metabolism

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Collection: 01-internacional Database: MEDLINE Main subject: Connexin 43 / Heart Conduction System / Heart Ventricles Limits: Animals Language: En Journal: Circulation Year: 2001 Type: Article Affiliation country: United States

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