A functional switch from lung cancer resistance to susceptibility at the Pas1 locus in Kras2LA2 mice.

To, Minh D; Perez-Losada, Jesus; Mao, Jian-Hua; Hsu, Jeff; Jacks, Tyler; Balmain, Allan

To, Minh D; Perez-Losada, Jesus; Mao, Jian-Hua; Hsu, Jeff; Jacks, Tyler; Balmain, Allan.

Affiliation

To MD; University of California San Francisco (UCSF) Comprehensive Cancer Center, San Francisco, California 94115, USA.

Nat Genet ; 38(8): 926-30, 2006 Aug.

Article in En | MEDLINE | ID: mdl-16823377

Subject(s)

Lung Neoplasms/genetics; Proto-Oncogene Proteins p21(ras)/genetics; Adenoma/chemically induced; Adenoma/genetics; Alleles; Animals; Carcinogens/toxicity; Female; Genetic Predisposition to Disease; Lung Neoplasms/chemically induced; Male; Mice; Models, Genetic; Mutation; Oncogenes; Risk Factors; Urethane/toxicity

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Proto-Oncogene Proteins p21(ras) / Lung Neoplasms Type of study: Etiology_studies / Prognostic_studies / Risk_factors_studies Limits: Animals Language: En Journal: Nat Genet Journal subject: GENETICA MEDICA Year: 2006 Type: Article Affiliation country: United States

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