Ras activity levels control the development of pancreatic diseases.

Ji, Baoan; Tsou, Lilian; Wang, Huamin; Gaiser, Sebastian; Chang, David Z; Daniluk, Jaroslaw; Bi, Yan; Grote, Tobias; Longnecker, Daniel S; Logsdon, Craig D

Ji, Baoan; Tsou, Lilian; Wang, Huamin; Gaiser, Sebastian; Chang, David Z; Daniluk, Jaroslaw; Bi, Yan; Grote, Tobias; Longnecker, Daniel S; Logsdon, Craig D.

Affiliation

Ji B; Department of Cancer Biology, University of Texas M. D. Anderson Cancer Center, Houston, Texas 77030, USA.

Gastroenterology ; 137(3): 1072-82, 1082.e1-6, 2009 Sep.

Article in En | MEDLINE | ID: mdl-19501586

Subject(s)

Carcinoma, Pancreatic Ductal/metabolism; Genes, ras/physiology; Pancreatic Neoplasms/metabolism; Pancreatitis, Chronic/metabolism; Animals; Carcinoma, Pancreatic Ductal/genetics; Carcinoma, Pancreatic Ductal/pathology; Cell Line, Tumor; Genes, ras/genetics; Humans; Mice; Mice, Transgenic; Mutation; Pancreatic Neoplasms/genetics; Pancreatic Neoplasms/pathology; Pancreatitis, Chronic/genetics; Pancreatitis, Chronic/pathology

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Pancreatic Neoplasms / Genes, ras / Carcinoma, Pancreatic Ductal / Pancreatitis, Chronic Limits: Animals / Humans Language: En Journal: Gastroenterology Year: 2009 Type: Article Affiliation country: United States

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