Only a subset of Met-activated pathways are required to sustain oncogene addiction.

Bertotti, Andrea; Burbridge, Mike F; Gastaldi, Stefania; Galimi, Francesco; Torti, Davide; Medico, Enzo; Giordano, Silvia; Corso, Simona; Rolland-Valognes, Gaëlle; Lockhart, Brian P; Hickman, John A; Comoglio, Paolo M; Trusolino, Livio

Bertotti, Andrea; Burbridge, Mike F; Gastaldi, Stefania; Galimi, Francesco; Torti, Davide; Medico, Enzo; Giordano, Silvia; Corso, Simona; Rolland-Valognes, Gaëlle; Lockhart, Brian P; Hickman, John A; Comoglio, Paolo M; Trusolino, Livio.

Affiliation

Bertotti A; Division of Molecular Oncology, Institute for Cancer Research and Treatment (IRCC), University of Torino Medical School, 10060 Candiolo (Torino), Italy.

Sci Signal ; 2(102): er11, 2009.

Article in En | MEDLINE | ID: mdl-20039471

Subject(s)

Cell Cycle/physiology; Gene Silencing/physiology; Neoplasms/metabolism; Oncogenes/physiology; Proto-Oncogene Proteins c-met/metabolism; Receptors, Growth Factor/metabolism; Signal Transduction/physiology; Blotting, Western; Cell Line, Tumor; ErbB Receptors/antagonists & inhibitors; Gene Expression Profiling; Green Fluorescent Proteins/metabolism; Humans; Oligonucleotide Array Sequence Analysis; Oncogene Protein p21(ras)/metabolism; Phosphatidylinositol 3-Kinase/metabolism; Polymerase Chain Reaction; Proteomics; Proto-Oncogene Proteins c-met/antagonists & inhibitors; Proto-Oncogene Proteins c-met/genetics; Receptors, Growth Factor/antagonists & inhibitors; Receptors, Growth Factor/genetics; Sequence Analysis, DNA

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Collection: 01-internacional Database: MEDLINE Main subject: Oncogenes / Signal Transduction / Cell Cycle / Receptors, Growth Factor / Proto-Oncogene Proteins c-met / Gene Silencing / Neoplasms Type of study: Prognostic_studies Limits: Humans Language: En Journal: Sci Signal Journal subject: CIENCIA / FISIOLOGIA Year: 2009 Type: Article Affiliation country: Italy

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