Amyloid-ß peptide-induced extracellular S100A9 depletion is associated with decrease of antimicrobial peptide activity in human THP-1 monocytes.

Lee, Eun Ok; Yang, Ji Hye; Chang, Keun-A; Suh, Yoo-Hun; Chong, Young Hae

Lee, Eun Ok; Yang, Ji Hye; Chang, Keun-A; Suh, Yoo-Hun; Chong, Young Hae.

Affiliation

Lee EO; Department of Microbiology, School of Medicine, Ewha Medical Research Institute, Ewha Womans University, 911-1, Mok-6-dong, Yangcheonku, Seoul 158-710, Republic of Korea.

J Neuroinflammation ; 10: 68, 2013 May 30.

Article in En | MEDLINE | ID: mdl-23721320

Subject(s)

Amyloid beta-Peptides/toxicity; Antimicrobial Cationic Peptides/biosynthesis; Calgranulin B/metabolism; Monocytes/metabolism; Blotting, Western; Calcium/metabolism; Cells, Cultured; Colony-Forming Units Assay; Electrophoresis, Polyacrylamide Gel; Escherichia coli/genetics; Escherichia coli/metabolism; Humans; Immunity, Innate/drug effects; Microscopy, Fluorescence; Monocytes/drug effects; RNA, Small Interfering/genetics; Recombinant Proteins/metabolism; Tetrazolium Salts; Thiazoles

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Monocytes / Amyloid beta-Peptides / Antimicrobial Cationic Peptides / Calgranulin B Type of study: Risk_factors_studies Limits: Humans Language: En Journal: J Neuroinflammation Journal subject: NEUROLOGIA Year: 2013 Type: Article

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