Loss of HDAC11 ameliorates clinical symptoms in a multiple sclerosis mouse model.

Sun, Lei; Telles, Elphine; Karl, Molly; Cheng, Fengdong; Luetteke, Noreen; Sotomayor, Eduardo M; Miller, Robert H; Seto, Edward

Sun, Lei; Telles, Elphine; Karl, Molly; Cheng, Fengdong; Luetteke, Noreen; Sotomayor, Eduardo M; Miller, Robert H; Seto, Edward.

Affiliation

Sun L; George Washington University Cancer Center, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Telles E; Department of Biochemistry and Molecular Medicine, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Karl M; George Washington University Cancer Center, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Cheng F; Department of Anatomy and Cell Biology, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Luetteke N; George Washington University Cancer Center, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Sotomayor EM; George Washington University Cancer Center, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Miller RH; George Washington University Cancer Center, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.
Seto E; Department of Anatomy and Cell Biology, George Washington University School of Medicine and Health Sciences, Washington, DC, USA.

Life Sci Alliance ; 1(5): e201800039, 2018 Oct.

Article in En | MEDLINE | ID: mdl-30456376

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Full text: 1 Collection: 01-internacional Database: MEDLINE Type of study: Diagnostic_studies / Prognostic_studies Language: En Journal: Life Sci Alliance Year: 2018 Type: Article Affiliation country: United States

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