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Targeted genomic deletions identify diverse enhancer functions and generate a kidney-specific, endocrine-deficient Cyp27b1 pseudo-null mouse.
Meyer, Mark B; Benkusky, Nancy A; Kaufmann, Martin; Lee, Seong Min; Redfield, Robert R; Jones, Glenville; Pike, J Wesley.
Affiliation
  • Meyer MB; From the Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706, markmeyer@wisc.edu.
  • Benkusky NA; From the Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
  • Kaufmann M; the Departments of Biomedical and Molecular Sciences and.
  • Lee SM; Surgery, Queen's University, Kingston, Ontario K7L 3N6, Canada, and.
  • Redfield RR; From the Department of Biochemistry, University of Wisconsin, Madison, Wisconsin 53706.
  • Jones G; the Department of Surgery, University of Wisconsin School of Medicine and Public Health, Madison, Wisconsin 53706.
  • Pike JW; the Departments of Biomedical and Molecular Sciences and.
J Biol Chem ; 294(24): 9518-9535, 2019 06 14.
Article in En | MEDLINE | ID: mdl-31053643
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Vitamin D / Calcium / Enhancer Elements, Genetic / Gene Deletion / Homeostasis / 25-Hydroxyvitamin D3 1-alpha-Hydroxylase / Kidney Type of study: Prognostic_studies Limits: Animals / Female / Humans / Male Language: En Journal: J Biol Chem Year: 2019 Type: Article
Fulltext
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PubMed Links
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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Vitamin D / Calcium / Enhancer Elements, Genetic / Gene Deletion / Homeostasis / 25-Hydroxyvitamin D3 1-alpha-Hydroxylase / Kidney Type of study: Prognostic_studies Limits: Animals / Female / Humans / Male Language: En Journal: J Biol Chem Year: 2019 Type: Article