Regulation of Tau protein phosphorylation by glucosamine-induced O-GlcNAcylation as a neuroprotective mechanism in a brain ischemia-reperfusion model.

Cardozo, C F; Vera, A; Quintana-Peña, V; Arango-Davila, C A; Rengifo, J

Cardozo, C F; Vera, A; Quintana-Peña, V; Arango-Davila, C A; Rengifo, J.

Affiliation

Cardozo CF; Escuela de Ciencias Básicas, Universidad del Valle, Cali, Colombia.
Vera A; Facultad de Ciencias Naturales, Universidad Icesi, Cali, Colombia.
Quintana-Peña V; Departamento de Ciencias Básicas, Universidad de Caldas, Manizales, Colombia.
Arango-Davila CA; Facultad de Ciencias de la Salud, Universidad Icesi, Cali, Colombia.
Rengifo J; Facultad de Ciencias de la Salud, Universidad Icesi, Cali, Colombia.

Int J Neurosci ; 133(2): 194-200, 2023 Feb.

Article in En | MEDLINE | ID: mdl-33736564

Subject(s)

Brain Ischemia; Neuroprotective Agents; Reperfusion Injury; Rats; Animals; Male; Glucosamine/pharmacology; tau Proteins/metabolism; Phosphorylation; Rats, Wistar; Brain Ischemia/drug therapy; Ischemia; Reperfusion; Reperfusion Injury/drug therapy; Reperfusion Injury/metabolism; Neuroprotective Agents/pharmacology

Key words

Brain ischemia; O-GlcNAc; Tau phosphorylation; glucosamine; neuroprotection

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Full text: 1 Collection: 01-internacional Database: MEDLINE Main subject: Reperfusion Injury / Brain Ischemia / Neuroprotective Agents Type of study: Clinical_trials Limits: Animals Language: En Journal: Int J Neurosci Year: 2023 Type: Article Affiliation country: Colombia

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