Lack of cognate help by CD4+ T cells and anergy of CD8+ T cells are the principal mechanisms for anti-leukocyte function-associated antigen-1/intercellular adhesion molecule-1-induced cardiac allograft tolerance.

Bashuda, H; Seino, K; Ra, C; Yagita, H; Okumura, K

Bashuda, H; Seino, K; Ra, C; Yagita, H; Okumura, K.

Affiliation

Bashuda H; Department of Immunology, Juntendo University School of Medicine, Bunkyo-ku, Tokyo, Japan.

Transplantation ; 63(1): 113-8, 1997 Jan 15.

Article in En | MEDLINE | ID: mdl-9000671

Subject(s)

CD4-Positive T-Lymphocytes/immunology; CD8-Positive T-Lymphocytes/immunology; Heart Transplantation/immunology; Immune Tolerance; Intercellular Adhesion Molecule-1/physiology; Lymphocyte Function-Associated Antigen-1/physiology; Animals; Graft Survival; Isoantigens/immunology; Lymphocyte Activation; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C3H; Mice, Inbred C57BL; Skin Transplantation; T-Lymphocytes, Cytotoxic/physiology

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Collection: 01-internacional Database: MEDLINE Main subject: CD4-Positive T-Lymphocytes / Lymphocyte Function-Associated Antigen-1 / Heart Transplantation / Intercellular Adhesion Molecule-1 / CD8-Positive T-Lymphocytes / Immune Tolerance Type of study: Risk_factors_studies Limits: Animals Language: En Journal: Transplantation Year: 1997 Type: Article Affiliation country: Japan

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