Suppression of glypican-1 autodegradation by NO-deprivation correlates with nuclear accumulation of amyloid beta in normal fibroblasts.

Cheng, Fang; Fransson, Lars-Åke; Mani, Katrin

Cheng, Fang; Fransson, Lars-Åke; Mani, Katrin.

Afiliación

Cheng F; Department of Experimental Medical Science, Division of Neuroscience, Glycobiology Group, Lund University, Biomedical Center A13, SE-221 84, Lund, Sweden.
Fransson LÅ; Department of Experimental Medical Science, Division of Neuroscience, Glycobiology Group, Lund University, Biomedical Center A13, SE-221 84, Lund, Sweden.
Mani K; Department of Experimental Medical Science, Division of Neuroscience, Glycobiology Group, Lund University, Biomedical Center A13, SE-221 84, Lund, Sweden. katrin.mani@med.lu.se.

Glycoconj J ; 32(9): 675-84, 2015 Dec.

Article en En | MEDLINE | ID: mdl-26318599

Asunto(s)

Péptidos beta-Amiloides/metabolismo; Fibroblastos/metabolismo; Glipicanos/metabolismo; Óxido Nítrico/deficiencia; Proteolisis; Animales; Línea Celular; Núcleo Celular/metabolismo; Ratones; Óxido Nítrico/metabolismo

Palabras clave

Alzheimer disease; Amyloid beta; Glypican-1; Heparan sulfate; Nitric oxide

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Péptidos beta-Amiloides / Glipicanos / Proteolisis / Fibroblastos / Óxido Nítrico Límite: Animals Idioma: En Revista: Glycoconj J Asunto de la revista: BIOQUIMICA / METABOLISMO Año: 2015 Tipo del documento: Article País de afiliación: Suecia

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