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Reduced supply of monocyte-derived macrophages leads to a transition from nodular to diffuse lesions and tissue cell activation in silica-induced pulmonary fibrosis in mice.
Shichino, Shigeyuki; Abe, Jun; Ueha, Satoshi; Otsuji, Mikiya; Tsukui, Tatsuya; Kosugi-Kanaya, Mizuha; Shand, Francis H W; Hashimoto, Shin-Ichi; Suzuki, Hiroshi I; Morikawa, Teppei; Inagaki, Yutaka; Matsushima, Kouji.
Afiliación
  • Shichino S; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Abe J; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Ueha S; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Otsuji M; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Tsukui T; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Kosugi-Kanaya M; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan.
  • Shand FH; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Department of Pharmacology and Therapeutics, the University of Melbourne, Parkville, Australia.
  • Hashimoto S; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan; Division of Nephrology, Department of Laboratory Medicine, Kanazawa Univer
  • Suzuki HI; Department of Molecular Pathology, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan.
  • Morikawa T; Department of Pathology, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan.
  • Inagaki Y; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan; Center for Matrix Biology and Medicine, Tokai University, Kanagawa, Japan.
  • Matsushima K; Department of Molecular Preventive Medicine, Graduate School of Medicine, the University of Tokyo, Tokyo, Japan; Japan Science and Technology Agency, Core Research for Evolutional Science and Technology (CREST), Tokyo, Japan. Electronic address: koujim@m.u-tokyo.ac.jp.
Am J Pathol ; 185(11): 2923-38, 2015 Nov.
Article en En | MEDLINE | ID: mdl-26456580
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Macrófagos Alveolares / Receptores CCR2 Límite: Animals / Humans Idioma: En Revista: Am J Pathol Año: 2015 Tipo del documento: Article País de afiliación: Japón
Texto completo
Imprimir
XML
PubMed Links
Buscar en Google

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Fibrosis Pulmonar / Macrófagos Alveolares / Receptores CCR2 Límite: Animals / Humans Idioma: En Revista: Am J Pathol Año: 2015 Tipo del documento: Article País de afiliación: Japón