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Activation of the IGF1 pathway mediates changes in cellular contractility and motility in single-suture craniosynostosis.
Al-Rekabi, Zeinab; Wheeler, Marsha M; Leonard, Andrea; Fura, Adriane M; Juhlin, Ilsa; Frazar, Christopher; Smith, Joshua D; Park, Sarah S; Gustafson, Jennifer A; Clarke, Christine M; Cunningham, Michael L; Sniadecki, Nathan J.
Afiliación
  • Al-Rekabi Z; Department of Mechanical Engineering, University of Washington, Seattle, WA 98195, USA Seattle Children's Research Institute, Center for Developmental Biology and Regenerative Medicine, Seattle, WA 98101, USA.
  • Wheeler MM; Department of Genome Sciences, University of Washington, Seattle, WA 98105, USA.
  • Leonard A; Department of Mechanical Engineering, University of Washington, Seattle, WA 98195, USA.
  • Fura AM; Department of Bioengineering, University of Washington, Seattle, WA 98105, USA.
  • Juhlin I; Department of Mechanical Engineering, University of Washington, Seattle, WA 98195, USA.
  • Frazar C; Department of Genome Sciences, University of Washington, Seattle, WA 98105, USA.
  • Smith JD; Department of Genome Sciences, University of Washington, Seattle, WA 98105, USA.
  • Park SS; Seattle Children's Research Institute, Center for Developmental Biology and Regenerative Medicine, Seattle, WA 98101, USA.
  • Gustafson JA; Seattle Children's Research Institute, Center for Developmental Biology and Regenerative Medicine, Seattle, WA 98101, USA.
  • Clarke CM; Seattle Children's Research Institute, Center for Developmental Biology and Regenerative Medicine, Seattle, WA 98101, USA.
  • Cunningham ML; Seattle Children's Research Institute, Center for Developmental Biology and Regenerative Medicine, Seattle, WA 98101, USA Division of Craniofacial Medicine and the Department of Pediatrics, University of Washington, Seattle, WA 98105, USA.
  • Sniadecki NJ; Department of Mechanical Engineering, University of Washington, Seattle, WA 98195, USA Department of Bioengineering, University of Washington, Seattle, WA 98105, USA nsniadec@uw.edu.
J Cell Sci ; 129(3): 483-91, 2016 Feb 01.
Article en En | MEDLINE | ID: mdl-26659664

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Factor I del Crecimiento Similar a la Insulina / Transducción de Señal Límite: Humans Idioma: En Revista: J Cell Sci Año: 2016 Tipo del documento: Article País de afiliación: Estados Unidos

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Factor I del Crecimiento Similar a la Insulina / Transducción de Señal Límite: Humans Idioma: En Revista: J Cell Sci Año: 2016 Tipo del documento: Article País de afiliación: Estados Unidos