Progression of chronic kidney disease: too much cellular talk causes damage.

Gewin, Leslie; Zent, Roy; Pozzi, Ambra

Gewin, Leslie; Zent, Roy; Pozzi, Ambra.

Afiliación

Gewin L; Division of Nephrology, Department of Medicine, Vanderbilt Medical Center, Nashville, Tennessee, USA; Department of Cell and Developmental Biology, Vanderbilt Medical Center, Nashville, Tennessee, USA; Veterans Affairs Medical Center, Nashville, Tennessee, USA.
Zent R; Division of Nephrology, Department of Medicine, Vanderbilt Medical Center, Nashville, Tennessee, USA; Department of Cell and Developmental Biology, Vanderbilt Medical Center, Nashville, Tennessee, USA; Veterans Affairs Medical Center, Nashville, Tennessee, USA; Department of Cancer Biology, Vanderbilt Medical Center, Nashville, Tennessee, USA. Electronic address: roy.zent@vanderbilt.edu.
Pozzi A; Division of Nephrology, Department of Medicine, Vanderbilt Medical Center, Nashville, Tennessee, USA; Veterans Affairs Medical Center, Nashville, Tennessee, USA; Department of Cancer Biology, Vanderbilt Medical Center, Nashville, Tennessee, USA; Department of Molecular Physiology and Biophysics, Vanderbilt Medical Center, Nashville, Tennessee, USA.

Kidney Int ; 91(3): 552-560, 2017 03.

Article en En | MEDLINE | ID: mdl-27773427

Asunto(s)

Comunicación Celular; Citocinas/metabolismo; Riñón/metabolismo; Insuficiencia Renal Crónica/metabolismo; Transducción de Señal; Animales; Citocinas/inmunología; Fibrosis; Humanos; Riñón/inmunología; Riñón/patología; Insuficiencia Renal Crónica/inmunología; Insuficiencia Renal Crónica/patología

Palabras clave

endothelial cells; epithelial cells; fibroblasts; fibrosis; inflammation; interstitium

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Transducción de Señal / Comunicación Celular / Citocinas / Insuficiencia Renal Crónica / Riñón Tipo de estudio: Etiology_studies Límite: Animals / Humans Idioma: En Revista: Kidney Int Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos

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