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Myt1l safeguards neuronal identity by actively repressing many non-neuronal fates.
Mall, Moritz; Kareta, Michael S; Chanda, Soham; Ahlenius, Henrik; Perotti, Nicholas; Zhou, Bo; Grieder, Sarah D; Ge, Xuecai; Drake, Sienna; Euong Ang, Cheen; Walker, Brandon M; Vierbuchen, Thomas; Fuentes, Daniel R; Brennecke, Philip; Nitta, Kazuhiro R; Jolma, Arttu; Steinmetz, Lars M; Taipale, Jussi; Südhof, Thomas C; Wernig, Marius.
Afiliación
  • Mall M; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Kareta MS; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Chanda S; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Ahlenius H; Department of Molecular and Cellular Physiology and Howard Hughes Medical Institute, Stanford University, Stanford, California 94305, USA.
  • Perotti N; Department of Clinical Sciences, Division of Neurology and Lund Stem Cell Center, Lund University, 221 84 Lund, Sweden.
  • Zhou B; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Grieder SD; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Ge X; Department of Molecular and Cellular Physiology and Howard Hughes Medical Institute, Stanford University, Stanford, California 94305, USA.
  • Drake S; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Euong Ang C; Department of Developmental Biology, Stanford University, Stanford, California 94305, USA.
  • Walker BM; Department of Clinical Sciences, Division of Neurology and Lund Stem Cell Center, Lund University, 221 84 Lund, Sweden.
  • Vierbuchen T; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Fuentes DR; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Brennecke P; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Nitta KR; Department of Pathology and Institute for Stem Cell Biology and Regenerative Medicine, Stanford University, Stanford, California 94305, USA.
  • Jolma A; Department of Genetics, Stanford University, Stanford, California 94305, USA.
  • Steinmetz LM; Division of Functional Genomics and Systems Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, 171 77 Stockholm, Sweden.
  • Taipale J; Division of Functional Genomics and Systems Biology, Department of Medical Biochemistry and Biophysics, Karolinska Institutet, 171 77 Stockholm, Sweden.
  • Südhof TC; Department of Genetics, Stanford University, Stanford, California 94305, USA.
  • Wernig M; Genome Biology Unit, European Molecular Biology Laboratory (EMBL), 69117 Heidelberg, Germany.
Nature ; 544(7649): 245-249, 2017 04 13.
Article en En | MEDLINE | ID: mdl-28379941
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Proteínas Represoras / Factores de Transcripción / Linaje de la Célula / Silenciador del Gen / Reprogramación Celular / Neurogénesis / Proteínas del Tejido Nervioso / Neuronas Límite: Animals / Humans Idioma: En Revista: Nature Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Proteínas Represoras / Factores de Transcripción / Linaje de la Célula / Silenciador del Gen / Reprogramación Celular / Neurogénesis / Proteínas del Tejido Nervioso / Neuronas Límite: Animals / Humans Idioma: En Revista: Nature Año: 2017 Tipo del documento: Article País de afiliación: Estados Unidos