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The chemokine receptor CCR10 promotes inflammation-driven hepatocarcinogenesis via PI3K/Akt pathway activation.
Wu, Qiong; Chen, Jin-Xian; Chen, Yu; Cai, Li-Li; Wang, Xiao-Zhong; Guo, Wu-Hua; Zheng, Jian-Feng.
Afiliación
  • Wu Q; Department of Clinical Laboratory Medicine, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
  • Chen JX; Department of Gastrointestinal Surgery, Renji Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, 200127, China.
  • Chen Y; Department of Clinical Laboratory Medicine, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
  • Cai LL; Department of Clinical Laboratory Medicine, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
  • Wang XZ; Department of Clinical Laboratory Medicine, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China.
  • Guo WH; Department of Gastroenterology, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China. guowuhua@aliyun.com.
  • Zheng JF; Department of Clinical Laboratory Medicine, The Second Affiliated Hospital of Nanchang University, Nanchang, 330006, China. jfzheng66@163.com.
Cell Death Dis ; 9(2): 232, 2018 02 14.
Article en En | MEDLINE | ID: mdl-29445190

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Regulación Neoplásica de la Expresión Génica / Carcinoma Hepatocelular / Fosfatidilinositol 3-Quinasas / Receptores CCR10 / Hemangioma / Neoplasias Hepáticas / Neoplasias Hepáticas Experimentales Tipo de estudio: Prognostic_studies Idioma: En Revista: Cell Death Dis Año: 2018 Tipo del documento: Article País de afiliación: China

Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Regulación Neoplásica de la Expresión Génica / Carcinoma Hepatocelular / Fosfatidilinositol 3-Quinasas / Receptores CCR10 / Hemangioma / Neoplasias Hepáticas / Neoplasias Hepáticas Experimentales Tipo de estudio: Prognostic_studies Idioma: En Revista: Cell Death Dis Año: 2018 Tipo del documento: Article País de afiliación: China