Receptor-Interacting Protein 140 Enhanced Temozolomide-Induced Cellular Apoptosis Through Regulation of E2F1 in Human Glioma Cell Lines.

Tsai, Hong-Chieh; Wei, Kuo-Chen; Chen, Pin-Yuan; Huang, Chiung-Yin; Chen, Ko-Ting; Lin, Ya-Jui; Cheng, Hsiao-Wei; Huang, Chun-Hao; Wang, Hsiang-Tsui

Tsai, Hong-Chieh; Wei, Kuo-Chen; Chen, Pin-Yuan; Huang, Chiung-Yin; Chen, Ko-Ting; Lin, Ya-Jui; Cheng, Hsiao-Wei; Huang, Chun-Hao; Wang, Hsiang-Tsui.

Afiliación

Tsai HC; Department of Neurosurgery, Linkou Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan.
Wei KC; Graduate Institute of Clinical Medical Sciences and School of Traditional Chinese Medicine, Chang Gung University, Taoyuan, 333, Taiwan.
Chen PY; Department of Neurosurgery, New Taipei Municipal TuCheng Hospital, Chang Gung Memorial Hospital, New Taipei Municipal, Taipei, 236, Taiwan.
Huang CY; Department of Neurosurgery, Keelung Chang Gung Memorial Hospital, Keelung, 204, Taiwan.
Chen KT; Department of Neurosurgery, Keelung Chang Gung Memorial Hospital, Keelung, 204, Taiwan.
Lin YJ; School of Medicine, Chang Gung University, Taoyuan, 333, Taiwan.
Cheng HW; Department of Neurosurgery, Linkou Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan.
Huang CH; School of Medicine, Chang Gung University, Taoyuan, 333, Taiwan.
Wang HT; Department of Neurosurgery, Linkou Chang Gung Memorial Hospital, Taoyuan, 333, Taiwan.

Neuromolecular Med ; 24(2): 113-124, 2022 06.

Article en En | MEDLINE | ID: mdl-34075570

Asunto(s)

Neoplasias Encefálicas; Glioblastoma; Glioma; Co-Represor 1 de Receptor Nuclear; Temozolomida; Apoptosis; Neoplasias Encefálicas/tratamiento farmacológico; Neoplasias Encefálicas/genética; Línea Celular Tumoral; Resistencia a Antineoplásicos; Factor de Transcripción E2F1/genética; Factor de Transcripción E2F1/metabolismo; Regulación Neoplásica de la Expresión Génica; Glioblastoma/tratamiento farmacológico; Glioblastoma/genética; Glioma/tratamiento farmacológico; Glioma/genética; Humanos; Co-Represor 1 de Receptor Nuclear/genética; Co-Represor 1 de Receptor Nuclear/metabolismo; Proteína de Interacción con Receptores Nucleares 1; Temozolomida/farmacología

Palabras clave

Apoptosis; E2F1; Glioblastoma multiforme; Receptor-interacting protein 140; Temozolomide

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Neoplasias Encefálicas / Glioblastoma / Co-Represor 1 de Receptor Nuclear / Temozolomida / Glioma Tipo de estudio: Prognostic_studies Límite: Humans Idioma: En Revista: Neuromolecular Med Asunto de la revista: BIOLOGIA MOLECULAR / NEUROLOGIA Año: 2022 Tipo del documento: Article País de afiliación: Taiwán

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