Transthyretin deposition alters cardiomyocyte sarcomeric architecture, calcium transients, and contractile force.

Dittloff, Kyle T; Spanghero, Emanuele; Solís, Christopher; Banach, Kathrin; Russell, Brenda

Dittloff, Kyle T; Spanghero, Emanuele; Solís, Christopher; Banach, Kathrin; Russell, Brenda.

Afiliación

Dittloff KT; Department of Physiology and Biophysics, University of Illinois at Chicago, Chicago, Illinois, USA.
Spanghero E; Department of Biomedical Engineering, University of Illinois at Chicago, Chicago, Illinois, USA.
Solís C; Department of Physiology and Biophysics, University of Illinois at Chicago, Chicago, Illinois, USA.
Banach K; Department of Internal Medicine/Cardiology, Rush University Medical Center, Chicago, Illinois, USA.
Russell B; Department of Physiology and Biophysics, University of Illinois at Chicago, Chicago, Illinois, USA.

Physiol Rep ; 10(5): e15207, 2022 03.

Article en En | MEDLINE | ID: mdl-35262277

Asunto(s)

Neuropatías Amiloides Familiares; Insuficiencia Cardíaca; Animales; Calcio; Calcio de la Dieta; Ratones; Miocitos Cardíacos; Prealbúmina/química; Prealbúmina/farmacología; Ratas; Sarcómeros; Volumen Sistólico

Palabras clave

HFpEF; aging; amyloidosis; electromechanical uncoupling; heart failure; transthyretin

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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Neuropatías Amiloides Familiares / Insuficiencia Cardíaca Tipo de estudio: Prognostic_studies Límite: Animals Idioma: En Revista: Physiol Rep Año: 2022 Tipo del documento: Article País de afiliación: Estados Unidos

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