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Conserved reduction of m6A RNA modifications during aging and neurodegeneration is linked to changes in synaptic transcripts.
Castro-Hernández, Ricardo; Berulava, Tea; Metelova, Maria; Epple, Robert; Peña Centeno, Tonatiuh; Richter, Julia; Kaurani, Lalit; Pradhan, Ranjit; Sakib, M Sadman; Burkhardt, Susanne; Ninov, Momchil; Bohnsack, Katherine E; Bohnsack, Markus T; Delalle, Ivana; Fischer, Andre.
Afiliación
  • Castro-Hernández R; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Berulava T; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Metelova M; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Epple R; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Peña Centeno T; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Richter J; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Kaurani L; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Pradhan R; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Sakib MS; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Burkhardt S; Department for Epigenetics and Systems Medicine in Neurodegenerative Diseases, German Center for Neurodegenerative Diseases, 37077 Göttingen, Germany.
  • Ninov M; Department of Neurobiology, Max-Planck Institute for Biophysical Chemistry, 37077 Göttingen, Germany.
  • Bohnsack KE; Department of Molecular Biology, University Medical Center, 37077 Göttingen, Germany.
  • Bohnsack MT; Department of Molecular Biology, University Medical Center, 37077 Göttingen, Germany.
  • Delalle I; Multiscale Bioimaging Cluster of Excellence, University of Göttingen, 37077 Göttingen, Germany.
  • Fischer A; Department of Pathology, Lifespan Academic Medical Center, Brown University, 02912 Providence, Rhode Island.
Proc Natl Acad Sci U S A ; 120(9): e2204933120, 2023 02 28.
Article en En | MEDLINE | ID: mdl-36812208
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Proteína Quinasa Tipo 2 Dependiente de Calcio Calmodulina / Enfermedad de Alzheimer Límite: Aged / Animals / Humans Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2023 Tipo del documento: Article País de afiliación: Alemania
Texto completo
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Texto completo: 1 Colección: 01-internacional Banco de datos: MEDLINE Asunto principal: Proteína Quinasa Tipo 2 Dependiente de Calcio Calmodulina / Enfermedad de Alzheimer Límite: Aged / Animals / Humans Idioma: En Revista: Proc Natl Acad Sci U S A Año: 2023 Tipo del documento: Article País de afiliación: Alemania